The pathophysiology is similar to that seen in angina pectoris and intermittent claudication. The most common cause of abdominal angina is atherosclerotic vascular disease, where the occlusive process commonly involves the ostia and the proximal few centimeters of the mesenteric vessels. It can be associated with:
Hallmark of condition: Disabling midepigastric or central abdominal pain within 10–15 minutes after eating.
Physical examination: The abdomen typically is scaphoid and soft, even during an episode of pain. Patients present with stigmata of weight loss and signs of peripheral vascular disease, particularly aortoiliac occlusive disease, may be present.
Causes: Smoking is an associated risk factor. In most series, approximately 75-80% of patients smoke.
^Kapadia S, Parakh R, Grover T, Agarwal S (2005). "Side-to-side aorto-mesenteric anastomosis for management of abdominal angina". Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology24 (6): 256–7. PMID16424623.
^deVries H, Wijffels RT, Willemse PH et al. (2005). "Abdominal angina in patients with a midgut carcinoid, a sign of severe pathology". World journal of surgery29 (9): 1139–42. doi:10.1007/s00268-005-7825-x. PMID16086212.
^Choi BG, Jeon HS, Lee SO, Yoo WH, Lee ST, Ahn DS (2002). "Primary antiphospholipid syndrome presenting with abdominal angina and splenic infarction". Rheumatol. Int.22 (3): 119–21. doi:10.1007/s00296-002-0196-9. PMID12111088.
^Senechal Q, Massoni JM, Laurian C, Pernes JM (2001). "Transient relief of abdominal angina by Wallstent placement into an occluded superior mesenteric artery". The Journal of cardiovascular surgery42 (1): 101–5. PMID11292915.