George Perry (neuroscientist)

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George Perry
George-Perry.jpg
Born (1953-04-12) April 12, 1953 (age 61)
Lompoc, California
Residence San Antonio, Texas
Citizenship United States
Fields Neurology, Pathology
Institutions Baylor College of Medicine (1979-82), Case Western Reserve University (1982-present), University of Alaska (2001-present) and University of Texas at San Antonio (2006-present)
Alma mater Allan Hancock College (A.A., 1973), University of California at Santa Barbara (B.A., 1974), Hopkins Marine Station of Stanford University and Scripps Institution of Oceanography of University of California at San Diego (Ph.D., 1979)
Doctoral advisor David Epel
Other academic advisors Bill Brinkley, Joseph Bryan, Anthony R. Means and Pierluigi Gambetti
Notable students Paula Moreira, Mark Smith and Quan Liu
Known for Discoveries concerning neuronal oxidation of nucleic acids in Alzheimer's disease
Notable awards Denham Harman Research Award [American Aging Association], Alzheimer Award and Medal (twice)Journal of Alzheimer's Disease, ISI highly cited researcher, Iberoamerican Molecular Biology Organization, AAAS Fellow, Corresponding Member of Sciences Academy of Lisbon, Spanish Royal Academy of Sciences and the Mexican Academy of Sciences, SACNAS Distinguished Professional Mentor, member of the Dana Alliance, Senior Investigator Award International College of Geriatric Psychoneuropharmacology, Fellow of Linnean Society of London, Fellow of Microscopy Society of America, Fellow and Chartered Chemist of Royal Society of Chemistry, Fellow of Royal Society of Medicine, Fellow, Chartered Biologist and Chartered Scientist of Society of Biology,Fellow of Royal College of Pathologists, Sigma Xi, Phi Kappa Phi, Senior Fulbright Scholar, Panama National Plaque of Honor for Excellence in Neuroscience, and Sigma Xi Alamo Chapter Martin Goland Award.

George Perry (born April 12, 1953 in Lompoc, California) is a neuroscientist and Dean of the College of Sciences and Professor of Biology at the University of Texas at San Antonio. Perry is recognized in the field of Alzheimer's disease research particularly for his work on oxidative stress.

Education[edit]

Perry received his bachelor's of arts degree in Zoology from University of California, Santa Barbara. After graduation, he headed to Scripps Institution of Oceanography and also studied at Hopkins Marine Station of Stanford University and the Marine Biological Laboratory of Woods Hole and obtained his PhD in Marine Biology under David Epel in 1979. He then received a postdoctoral fellowship in the Department of Cell Biology in the laboratories of Drs. Bill Brinkley, Joseph Bryan and Anthony R. Means at Baylor College of Medicine where he laid the foundation for his observations of cytoskeletal abnormalities.

Professional Appointments[edit]

In 1995, Perry joined the faculty of Case Western Reserve University, where he currently holds an adjunct appointment. He is currently dean of the College of Sciences and professor of biology at the University of Texas at San Antonio. He is distinguished as one of the top Alzheimer’s disease researchers[1][2] with over 1000 publications, one of the top 100 most-cited scientists[3] in Neuroscience & Behavior and one of the top 25 scientists in free radical research.[4] Perry is highly cited (over 37,000 times;H=99;ISI/over 54,000 times;H=114;Google Scholar)and is recognized as an ISI highly cited researcher.[5] Perry is editor for numerous journals and is editor-in-chief for the Journal of Alzheimer's Disease. He is fellow of the American Association for the Advancement of Sciences,president of the Southwestern and Rocky Mountain Division of American Association for the Advancement of Sciences, Chair of the Board of the National Organization of Portuguese Americans and past-president of the American Association of Neuropathologists.

Research focus[edit]

Perry's research is primarily focused on the mechanism of formation and physiological consequences of the cytopathology of Alzheimer disease.[6] He has played a key role in elucidating oxidative damage as the initial cytopathological abnormality in Alzheimer disease. He is currently working to determine the sequence of events leading to neuronal oxidative damage and the source of the increased oxygen radicals. His current studies focus on two issues: (i) the metabolic basis for the mitochondrial damage restricted to vulnerable neurons; and (ii) the consequences of RNA oxidation on protein synthesis rate and fidelity.

References[edit]

External links[edit]