|Classification and external resources|
The diagnosis is by examination of tissue, e.g. a stomach biopsy.
- foveolar hyperplasia with gland tortuosity and dilation,
- smooth muscle hyperplasia in the lamina propria, and
- scant or minimal inflammation, i.e. lack of large numbers of neutrophils and plasma cells.
Reactive gastropathy has a large number of causes, including:
Relation to gastritis
As a reactive gastropathy may mimic a (true) gastritis symptomatically and visually in an endoscopic examination, it may incorrectly be referred to as a gastritis. Even aware of the underlying etiology of the pathologic process, e.g. NSAID use, the label "chemical gastritis" is applied to a chemical gastropathy. Etymologically, "gastritis", in this context, is a misnomer as the process is not predominantly inflammatory, i.e. the condition is not an -itis. This type of mislabeling is not uncommon in medicine. Steatohepatitis is another example of this; it is not a process with significant inflammation yet, confusingly, carries the -itis ending.
- Genta, RM. (Nov 2005). "Differential diagnosis of reactive gastropathy.". Semin Diagn Pathol 22 (4): 273–83. doi:10.1053/j.semdp.2006.04.001. PMID 16939055.
- Pashankar, DS.; Bishop, WP.; Mitros, FA. (Nov 2002). "Chemical gastropathy: a distinct histopathologic entity in children.". J Pediatr Gastroenterol Nutr 35 (5): 653–7. doi:10.1097/00005176-200211000-00012. PMID 12454581.
- Dixon, MF.; O'Connor, HJ.; Axon, AT.; King, RF.; Johnston, D. (May 1986). "Reflux gastritis: distinct histopathological entity?". J Clin Pathol 39 (5): 524–30. PMC 499914. PMID 3722405.