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When there's an excess of hepcidin (you have enough iron in your system) and an excess of iron passing the enterocytes in the intestine, how do enterocytes prevent too much iron entering their cytoplasma? — Preceding unsigned comment added by 18.104.22.168 (talk) 08:00, 4 June 2014 (UTC)
I know this straddles several areas but there is a fair amount of data on the gene and protein and less on the physiology from what I can tell so I think that the WP:MCBMOS style guide is must productive way to structure article.
My interest is from reading about erythropheresis in cancer but obviously iron homeostasis encompasses a lot more than that.