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According to this document It seems it was developed (co-developed?) at the ? (Institute of Organic Chemistry and biochemistry) in Prague. Is there some more reliable source for this?— Preceding unsigned comment added by Jirka6 (talk • contribs) March 7, 2007
"62% fewer infections in those taking Truvada and a 73% reduction in those who took a combination of Truvada and the HIV drug tenofovir."
-Truvada is combination of two drugs, emtricitabine and tenofovir. The second half of the sentence says something about taking a combination of Truvada AND "tenofovir," but tenofovir is already a component of Truvada. Are they really talking about taking Truvada and a little extra tenofovir on the side, or could this have been a mistake? — Preceding unsigned comment added by Arathur (talk • contribs) 02:18, 12 May 2012 (UTC)
Although Truvada alone can prevent an H.I.V. infection, it cannot control one that has already taken hold. If an H.I.V.-positive patient were to take Truvada alone, or if a user were to become infected while taking it, the H.I.V. strains they carry could become drug-resistant. The infection would be hard to treat, and it could spread to other people.
To prevent drug resistance, people who want to use Truvada for prevention first would have to make sure they were H.I.V.-negative and then be retested regularly, every few months, because it is possible, though unlikely, to become infected while taking the drug. But experts testifying before the panel on Thursday acknowledged that there is no way to ensure that people taking the drug receive frequent H.I.V. tests.
Then there is the question of adherence. Skipping doses or using Truvada on occasion, as if it were a party drug, would invite both infection and the emergence of drug-resistant strains. But research shows that people are not good at sticking to the required daily regimen. In one major study, only 10 percent of the participants took Truvada as directed — Preceding unsigned comment added by 184.108.40.206 (talk) 22:26, 16 July 2012 (UTC)