It is named after the physician, William Miller Ord, who first described it in 1877 and again in 1888.
It is more common among women than men.
Physiologically, antibodies to thyroid peroxidase and/or thyroglobulin cause gradual destruction of follicles in the thyroid gland. Accordingly, the disease can be detected clinically by looking for these antibodies in the blood. It is also characterised by invasion of the thyroid tissue by leukocytes, chiefly T-lymphocytes.
Ord's thyroiditis usually results in hypothyroidism.
Transient hyperthyroid states in the acute phase, (a common observation in Hashimoto's thyroiditis), are rare in Ord's disease.
This section is empty. You can help by adding to it. (October 2017)
Outside Europe a goitrous form of autoimmune thyroiditis (Hashimoto's Thyroiditis) is more common than Ord's disease.
- Davies, T. F. (2003), Ord-Hashimoto's Disease: Renaming a Common Disorder - Again. Thyroid 13 (4) 317. PMID 12820593.
- Williams D. (2003), Hashimoto's and Ord's diseases. Thyroid. 13(12): 1189. PMID 14751044