Talk:Sinoatrial node

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Untitled[edit]

Yes i have a question. Dose this sinoatrial react to pain, anger, or love and dose it know it.

Please anwser asap, and if you would like to see some of my work and class mate's work please go to Http://www.j00sux.com/templates/blackandgrey/index.php?user=language%20arts

Answer: Since the SA node is richly innervated with sympathetic fibers, it will react to any stimulus that can "activate" the sympathetic nervous system (fight-or-flight system). It could be activated by pain, anger, fright, ect. Think about this- any of these things could make your heart suddenly beat faster, and the SA node is the one controlling the beating. As far as "it" knowing what it does... it just reacts to chemical stimulus, as most things in the body do.

does thne SA work poorly if poor blood comes to the heart?

I have a better question. Why is it called the sinoatrial node? I understand the 'atrial' part but not the 'sino' or 'sinus' part of the name. And why isn't this included in the article? —Preceding unsigned comment added by Yahooshua (talk • contribs) 06:40, 14 September 2007 (UTC)[reply]

Suggestion for inclusion: The [sinoatrial node] imparts a distinct electrical signature to the commonly understood [EKG], representing the [PQ interval] or [P wave]. This signature is governed by [sympathetic] outflow in electrical polarization of the left and right atrial heart muscle. The robust signal from the [Sinoatrial Node] to the [AtrioVentricular Node] is then disseminated left and right in [Purkinge]s branches representing the [QRS] interval. Histologic examination of the sinoatrial and atrioventricular nodes suggests rich sympathetic influence within these miniscule cluster of cells. Electrical documentation of sympathetic influence of [Systole] is probably limited to the PQ and QRS intervals. The central body and skeleton of the heart [collagen] effectively block electrical influence between the atria and ventricle (barring WPW, LG, reentry circuits). The ST interval is posited as a [parasympathetic] EKG. Specificity in reading the systolic [PQRS] EKG is well documented. Nonspecific ST changes are poorly understood by most that routinely read EKGs. Computational dampening of sympathetic electrical outflow to illuminate parasympathetic inflow from the vagus and thoracic spinal accessory nerves to the SA node is likely to reveal why the SA node is also so richly parasympathetically innervated. Computational dampening of sympathetic electrical cardiac activity is likely to show a parasympathetic sump wallop delivered to the SA and AV nodes strongly suggesting that diastole is not a passive phase.--Lbeben (talk) 01:55, 19 July 2008 (UTC)[reply]

Graphics[edit]

Is there any reason that the same picture appears twice, right above each other? Is this 'site under constructin', that's what it looks like! --220.101.28.25 (talk) 12:33, 30 October 2009 (UTC)[reply]

Innervation[edit]

In my opinion the innervation of the sinoatrial node is applied by specific thoracic ganglia dedicated to individualized vagal branches. There is a method at work here. Leslie Beben

Someone needs to settle the argument in the innervation section, it's extremely confusing. 75.61.100.15 (talk) 04:10, 16 March 2010 (UTC)[reply]

Cite error: There are <ref> tags on this page without content in them (see the help page).== From where does SA Node gets electrical impulses / energy? ==

What is the source of SA node? OR Who supplies electricity to SA node? —Preceding unsigned comment added by 174.93.13.243 (talk) 18:44, 2 June 2010 (UTC)[reply]

I think, the SA node/area which receives impulse of foetus gets its first impulse from mother's body in the womb to start the cycle of both i.e. the blood supply and NERVE supply to whole body of a baby. turbo 10:27, 22 February 2022 (UTC) — Preceding unsigned comment added by Sidsandyy (talk • contribs) [reply]

Missing factors[edit]

Under - Clinical significance: Sinus node dysfunction describes an irregular heartbeat caused by faulty electrical signals of the heart.

This statement completely ignores the Vagus nerve's role in pacemaking, an extrinsic factor. It also doesn't mention the effective use of oral Theophylline to treat Sinus node dysfunction over the far more invasive implantation of a pacemaking device. For further information on these factors, see:

PubMed abstract - "Clinical characteristics of hypervagalotonic sinus node dysfunction", Korean J Intern Med. 2004 Sep;19(3):155-9, http://www.ncbi.nlm.nih.gov/pubmed/15481606

full and free KJIM article - "Clinical characteristics of hypervagalotonic sinus node dysfunction", Korean J Intern Med. 2004 Sep;19(3):155-9, http://www.kjim.org/journal/view.php?year=2004&vol=19&no=3&page=155

Thank you for your time, Wordreader (talk) 14:06, 25 July 2014 (UTC)[reply]

Proposal: move section[edit]

Under the Clinical significance section that discusses "sinus node dysfunction", the information should be moved to the relevant article on Sick sinus syndrome. After all, they are the same thing, are they not? Wordreader (talk) 14:15, 25 July 2014 (UTC)[reply]

Meaning of sinoatrial location and relationship to cardiac pacemaker[edit]

Hi Quercus solaris have reverted your edits on Sinoatrial node and no room to explain on page. Your first edits gave a different location and description in the lead that differed from that in the Location section (and all other refs). And just wrong to say it is chief component of cardiac pacemaker. Cheers --Iztwoz (talk) 23:24, 30 January 2016 (UTC)[reply]

Hi Iztwoz. Regarding the location, my edit was correct, but in different words from the way described in existing text and refs. The SVC enters the RA at the sinus venarum, and that's the very concept of where the word sinoatrial comes from and why the node that lies above that junction is even called the sinoatrial node. I think it's important to point out that my edit is not wrong, I just need refs to cite to support it. Regarding the relationship of the SA node to the cardiac pacemaker, there definitely is one, and the two articles definitely need to mention and link to each other in their ledes. Maybe the exact wording I used is not perfect, but it is a much bigger failing (in critical thinking) not to mention and link them at all, than to get the wording slightly wrong (which I may have done; I am not a cardiologist, and am still learning). I will find some wording that is better. Thanks, Quercus solaris (talk) 00:02, 31 January 2016 (UTC)[reply]

I made changes to address the concerns. Addressing this taught me about the crista terminalis atrii dextri, the terminal crest of the right atrium, where the venous sinus and atrium proper meet. I changed to say that the SA node is "the normal natural pacemaker of the heart", in line with what that article says about other natural pacemakers (ectopic ones). Quercus solaris (talk) 00:20, 31 January 2016 (UTC)[reply]

Does abbreviation "SAN" exist? I cant find it anywhere. Only SA node comes up in results. — Preceding unsigned comment added by 115.66.203.56 (talk) 15:30, 16 August 2017 (UTC)[reply]

Agree with you - SAN was added to page early in 2006. Shall remove it and see if anybody shouts. cheers --Iztwoz (talk) 15:40, 16 August 2017 (UTC)[reply]