Corticobasal syndrome: Difference between revisions
m Dating maintenance tags: {{Citation needed}} {{Empty section}} |
Afarzinkhan (talk | contribs) No edit summary |
||
| Line 21: | Line 21: | ||
__TOC__ |
__TOC__ |
||
== Signs and symptoms == |
== Signs and symptoms == |
||
Symptoms of CBS include [[apraxia]], alien limb phenomenon, frontal deficits, visuospatial deficits, acalculia, bradykinesia, dystonia, and [[Extrapyramidal symptoms|extrapyramidal]] motor symptoms such as [[myoclonus]] or rigidity.<ref name=Finger2016/> Movement deficits often begin on one side and progress to the other |
Symptoms of CBS include [[apraxia]], alien limb phenomenon, frontal deficits, visuospatial deficits, acalculia, bradykinesia, dystonia, and [[Extrapyramidal symptoms|extrapyramidal]] motor symptoms such as [[myoclonus]] or rigidity.<ref name=Finger2016/> Movement deficits often begin on one side and progress to the other<ref>{{cite journal |last1=Parmera |first1=Jacy Bezerra |last2=Rodriguez |first2=Roberta Dieh |last3=Studart Neto |first3=Adalberto |last4=Nitrini |first4=Ricardo |last5=Brucki |first5=Sonia Maria Dozzi |title=Corticobasal syndrome: A diagnostic conundrum |journal=Dementia & Neuropsychologia |date=2016 |volume=10 |issue=4 |pages=267–275 |doi=10.1590/s1980-5764-2016dn1004003 |url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5619264/#!po=8.62069 |issn=1980-5764}}</ref>.The symptoms of classic CBS differ from CBD in that CBD also features cognitive deficits in the [[executive functions]].<ref>{{Cite book|title=The Behavioral Neurology of Dementia|author=Fredericks CA, Lee SE |publisher=Cambridge University Press|year=2016|isbn=9781107077201|editor-last=Miller|editor-first=Bruce L.|edition=Second|location=Cambridge, United Kingdom|pages=203–6|chapter=The cognitive neurology of corticobasal degeneration and progressive supranuclear palsy|oclc=934020279|quote=[CBD is] reminiscent of classic CBS but with executive function deficits|editor-last2=Boeve|editor-first2=Bradley F.}}</ref> |
||
Also, intoxication with [[Annonaceae]] is believed to cause atypical forms of corticobasal syndromes.{{citation needed|date=December 2019}} |
Also, intoxication with [[Annonaceae]] is believed to cause atypical forms of corticobasal syndromes.{{citation needed|date=December 2019}} |
||
Revision as of 10:01, 16 January 2020
| Corticobasal syndrome | |
|---|---|
| Specialty | Neurology |
Corticobasal syndrome (CBS) is a progressive neurodegenerative disease and a type of frontotemporal dementia.[1] Corticobasal syndrome is one of four clinical phenotypes of corticobasal degeneration (CBD), the other three being: frontal behavioral-spatial syndrome (FBS), nonfluent/agrammatic variant of primary progressive aphasia (naPPA), and progressive supranuclear palsy syndrome (PSPS).[2]
CBD is the pathology underlying approximately 50% of CBS cases.[3] Other degenerative pathologies that can cause corticobasal syndrome include: Alzheimer's disease, Pick's disease with Pick bodies, progressive supranuclear palsy, Lewy body dementias, neurofilament inclusion body disease, Creutzfeldt-Jakob disease, frontotemporal degeneration due to progranulin gene mutation and motor neuron disease‐inclusion dementia.[4]
Signs and symptoms
Symptoms of CBS include apraxia, alien limb phenomenon, frontal deficits, visuospatial deficits, acalculia, bradykinesia, dystonia, and extrapyramidal motor symptoms such as myoclonus or rigidity.[1] Movement deficits often begin on one side and progress to the other[5].The symptoms of classic CBS differ from CBD in that CBD also features cognitive deficits in the executive functions.[6]
Also, intoxication with Annonaceae is believed to cause atypical forms of corticobasal syndromes.[citation needed]
Diagnosis
![[icon]](/wiki/File:Wiki_letter_w_cropped.svg){kind=small} | This section is empty. You can help by adding to it. (December 2019) |
References
- ^ a b Finger EC (April 2016). "Frontotemporal Dementias". Continuum (Minneap Minn) (Review). 22 (2 Dementia): 464–89. doi:10.1212/CON.0000000000000300. PMC 5390934. PMID 27042904.
- ^ Armstrong MJ, Litvan I, Lang AE, et al. (January 2013). "Criteria for the diagnosis of corticobasal degeneration". Neurology (Multicenter study). 80 (5): 496–503. doi:10.1212/WNL.0b013e31827f0fd1. PMC 3590050. PMID 23359374.
- ^ Gomperts SN (April 2016). "Lewy Body Dementias: Dementia With Lewy Bodies and Parkinson Disease Dementia". Continuum (Minneap Minn) (Review). 22 (2 Dementia): 435–63. doi:10.1212/CON.0000000000000309. PMC 5390937. PMID 27042903.
- ^ Hassan A, Whitwell JL, Josephs KA (November 2011). "The corticobasal syndrome-Alzheimer's disease conundrum". Expert Rev Neurother (Review). 11 (11): 1569–78. doi:10.1586/ern.11.153. PMC 3232678. PMID 22014136.
- ^ Parmera, Jacy Bezerra; Rodriguez, Roberta Dieh; Studart Neto, Adalberto; Nitrini, Ricardo; Brucki, Sonia Maria Dozzi (2016). "Corticobasal syndrome: A diagnostic conundrum". Dementia & Neuropsychologia. 10 (4): 267–275. doi:10.1590/s1980-5764-2016dn1004003. ISSN 1980-5764.
- ^ Fredericks CA, Lee SE (2016). "The cognitive neurology of corticobasal degeneration and progressive supranuclear palsy". In Miller, Bruce L.; Boeve, Bradley F. (eds.). The Behavioral Neurology of Dementia (Second ed.). Cambridge, United Kingdom: Cambridge University Press. pp. 203–6. ISBN 9781107077201. OCLC 934020279.
[CBD is] reminiscent of classic CBS but with executive function deficits