|WikiProject Molecular and Cellular Biology||(Rated Start-class, Top-importance)|
|A fact from Cell division appeared on Wikipedia's Main Page in the Did you know? column on 31 March 2004. The text of the entry was as follows: "Did you know Wikipedia:Recent additions/2004/March.||
If no one has the references given and can properly cite it reflect the new sources. Major edit possibly pending if citations for the current references isn't done by next week. (Psychro 06:30, 28 February 2007 (UTC))
Info from Cell (biology) to be included
The following info was dumped on Cell (biology). Please read it and see if it needs to be included in this article. I personally suspect it to be a description of mitosis. Striked through info was included in mitosis article after all. -- [[User:MacGyverMagic|Mgm|(talk)]] 08:52, Oct 21, 2004 (UTC)
Cell Division: The series of processes by which a cell divides into two or more cells. The stages of cell division: in order Cytokinesis: Cytoplasm divides. 2 daughter cells form.
Interphase: DNA is copied. Chromatin present. Centrioles present.
Prophase: Chromatin forms chromosomes. Nuclear membrane disappear.
Metaphase: Chromosomes are guided by centrioles and line up down the center of the cell.
Anaphase: Chromosomes split in half and start moving away from the center of cell.
Telephase: Chromosomes form chromatin again. 2 nuclear membranes form.
Daughter cells: the new cells formed by cell division
Chromatin: long tangled strands of DNA.
Chromosomes: DNA arranged in a neat “X” shape
Centrioles: The particles that move the organelles and DNA during cell division.
It should be noted that normal cells do indeed have a system to restore telomeres. As said, these wear out after some 52 divisions. A repair system is needed because otherwise cells would die in mere months. This is particularly true of lower intestine cells, for example.
The info on telomerase being absent in normal cells is wrong. Redmess 11:53, 7 May 2006 (UTC)
-I think it is not. Only stem cells still have telomerase. Differentiated cells don't => it's not possible to make infinite coltures with differentiated cells. --ITookUrNick 16:55, 24 May 2007 (UTC)
While you are editing this "Cells stop dividing because the telomeres, protective bits of DNA on the end of a chromosome." That is not a complete sentence. It is a preposition. You must add that the telomere do something at the end, or chamge it to read "of" the telomeres. TeigeRyan (talk) 08:35, 30 October 2009 (UTC)
Shouldn't this article be part of the above? Just asking... --Stormbay 18:00, 24 May 2007 (UTC)
I trhought that thyis gave iinformational things i would expiereement a little more Bill Nye the sience guy
- There's also sister taxa. It seems very biased toward women, but I'd say it was actually the opposite if anything. Ever notice how people always refer to an animal of unknown sex as 'he'? Richard001 (talk) 09:19, 21 August 2008 (UTC)
-Umm...no offence, but this 'talk' is irrevalent (pardon my spelling), and, yes, there is a none 'sexual' way of calling the offspring and that is calling it/them the 'offspring cells' a politically correct way of saying it, if you will. (And who's the jerk that put "WRONG"?) 188.8.131.52 (talk) 05:37, 3 December 2008 (UTC)
This is incorrect. I am not an expert on the subject, so I'm not going to elaborate that much - I'm just going to say that TELOMERASE is important in DNA REPLICATION and it is not only present in cancer cells - IT'S PRESENT IN ALL. It's used to elongate the lagging strand in the synthesis of DNA in order to prevent crucial proteins from being eliminated in the last stages of replication. Cancer cells do not stop growing because of inactivated tumor-suppressor genes (for example, p53 and pRb), not because of the presence of telomerase/telomeres. Someone should fix this. I couldn't figure out how to report an inaccurate article.
- The above comment was placed in the article at Cell division#Degradation by 184.108.40.206 at 04:03, 16 March 2011 (UTC). I have moved it to here and will inform the IP. Johnuniq (talk) 07:20, 16 March 2011 (UTC)