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This is an old revision of this page, as edited by 92.40.94.197 (talk) at 18:33, 29 August 2014 (→‎Pathophysiology theories). The present address (URL) is a permanent link to this revision, which may differ significantly from the current revision.

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Pathophysiology theories

Hey Lesion, received a response back from an endo colleague regarding the etiology of the pain after asking if it was a mild pulpitis,

With that info, I found a review at pubmed pmid 22238734. Will also post to talk:Toothache Ian Furst (talk) 11:31, 21 December 2013 (UTC)[reply]

Yes, this is what I was finding in the sources too. E.g.:


Therefore, I disagree with placing content about dentin hypersensitivity in a section called "pulpitis". If all pulpal causes of toothache to be merged into one section, it cannot be called "pulpitis" for accuracy... suggest "pulpal" instead. Lesion (talk) 12:15, 21 December 2013 (UTC)[reply]


Regarding reference 10 ( Schmidlin, Patrick R.; Sahrmann, Phlipp (30 December 2012). "Current management of dentin hypersensitivity". Clinical Oral Investigations 17 (S1): 55–59. doi:10.1007/s00784-012-0912-0.), I don't see how that article shows abrasive toothpaste contributes to the problem. — Preceding unsigned comment added by 32.217.10.85 (talk) 16:48, 29 August 2014 (UTC)[reply]

Not looked at the source in question, but abrasive toothpastes are a recognized cause of sensitvity. They cause tooth wear (technically, abrasion) with loss of tooth substance and resultant loss of thickness of enamel which "insulates" the dentin-pulp complex against stimulae. 92.40.94.197 (talk) 18:33, 29 August 2014 (UTC)[reply]