|Classification and external resources|
|ICD-10||G52.8, R51, G44.847|
Occipital neuralgia, also known as C2 neuralgia, or (rarely) Arnold's neuralgia, is a medical condition characterized by chronic pain in the upper neck, back of the head and behind the eyes. These areas correspond to the locations of the lesser and greater occipital nerves. The greater occipital nerve also has an artery that supplies blood that is wrapped around it - the occipital artery - that can contribute to the neuralgia. This condition is also sometimes characterized by diminished sensation in the affected area as well.
Occipital neuralgia is caused by damage to these nerves. Ways in which they can be damaged include trauma (usually concussive), physical stress on the nerve, repetitive neck contraction, flexion or extension, and as a result of medical complications (such as osteochondroma, a benign tumour of the bone). Another rare but possible cause is CSF Leaks. Yet another cause is from Radio Frequency nerve ablation. Rarely, occipital neuralgia may be a symptom of metastasis of certain cancers to the spine. There are several areas that have potential to cause injury from compression:
- The space between the vertebral bones of C1 and C2
- The atlantoaxial ligament as the dorsal ramus emerges
- The deep to superficial turn around the inferiolateral border of the obliquus capitis inferior muscle and its tight investing fascia
- The deep side of semispinalis capitis, where initial piercing can involve entrapment in either the muscle itself or surrounding fascia
- The superficial side of semispinalis capitis, where completion of nerve piercing muscle and its fascia again poses risk
6. The deep side of the trapezius as the nerve enters the muscle
- The tendinous insertion of the trapezius at the superior nuchal line
- The neurovascular intertwining of the GON and the occipital artery
The main symptom of this condition is chronic headache. The pain is commonly localized in the back and around or over the top of the head, sometimes up to the eyebrow or behind the eye. Because chronic headaches are a common symptom of numerous conditions, occipital neuralgia is often misdiagnosed at first, most commonly as tension headache or a migraine leading to unsuccessful treatment attempts. Another symptom is the eyes being sensitive to light, especially when headaches occur.
Occipital neuralgia is characterized by severe pain that begins in the upper neck and back of the head. This pain is typically one-sided, although it can be on both sides if both occipital nerves have been affected. Additionally, the pain may radiate forward toward the eye, as it follows the path of the occipital nerve(s). Individuals may notice blurred vision as the pain radiates near or behind the eye. The neuralgia pain is commonly described as sharp, shooting, zapping, an electric shock, or stabbing. The bouts of pain are rarely consistent, but can occur frequently with some patients depending on the damage to the nerves. The amount of time the pain lasts typically varies each time the symptom appears; it may last a few seconds or be almost continuous. Occipital neuralgia can last for hours or for several days.
Other symptoms of occipital neuralgia may include:
- Aching, burning, and throbbing pain that typically starts at the base of the head and radiates to the scalp
- Pain on one or both sides of the head
- Pain behind the eye
- Sensitivity to light
- Sensitivity to sound
- Slurred Speech
- Pain when moving the neck
- Difficulty with Balance and Coordination
- Tender scalp
- Nausea and/or vomiting
Once diagnosed, occipital neuralgia's symptoms can be treated/managed in several ways. There are a wide range of non-invasive treatments, including alternative treatments, which are as follows: acupuncture,chiropractic manipulation, occupational therapy, osteopathic manipulation,naprapathic treament, massage, yoga, physical therapy, rest, heat, anti- inflammatory medication, antidepressant medication, anti- convulsant medication, opioid and nonopioid analgesia, and migraine prophylaxis medication. Alternatives to these may include local nerve block, peripheral nerve stimulation, steroids, rhizotomy, phenol injections, antidepressants, and Occipital Cryoneurolysis.
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