Talk:Thrombus: Difference between revisions
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What treatents are there? Oral anticoagulants such as coumadin. Intravenous anticoagulants such as heparin, stretokinase, urokinase. Subcutaneous anticoagulants such as heparin, clexane. |
What treatents are there? Oral sex anticoagulants such as coumadin. Intravenous anticoagulants such as heparin, stretokinase, urokinase. Subcutaneous anticoagulants such as heparin, clexane. |
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A mural thrombus is a mass of dead cells, firbrin, and a few red cells. It does not embolize or liquify usually. You should not anticoagulate. |
A mural thrombus is a mass of dead cells, firbrin, and a few red cells. It does not embolize or liquify usually. You should not anticoagulate. |
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Revision as of 10:59, 6 January 2010
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Origin of the word
Anybody ?
What treatents are there? Oral sex anticoagulants such as coumadin. Intravenous anticoagulants such as heparin, stretokinase, urokinase. Subcutaneous anticoagulants such as heparin, clexane.
A mural thrombus is a mass of dead cells, firbrin, and a few red cells. It does not embolize or liquify usually. You should not anticoagulate.
cause of mural thrombus: almost always LAD thromboses. to treat LAD throbosis: 1. give aspirin (prevent another thrombosis of coronary vessel) 2. and warfarin/heparin (prevent mural thrombus formation)
mural thrombi = MIXED thrombi --> not pure venous or platelet. transmural infarction injures endothelial surface --> platelets stick.(tx=aspirin) next, ventricles not contracting well and get stasis --> venous like clot with coagulation factor involvement.(tx=heparin/warfarin) so, to prevent mural thrombus after infarct, give both aspirin and warfarin/heparin.