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{{student sandbox}}{{student sandbox}}'''Alcoholic hepatitis''' |
<ref>{{cite journal|doi=10.1016/0140-6736(90)90002-M}}</ref>{{student sandbox}}{{student sandbox}}'''Alcoholic hepatitis''' |
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Main article: [[Alcoholic hepatitis]] |
Main article: [[Alcoholic hepatitis]] |
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Revision as of 19:16, 24 October 2015
 | This is a user sandbox of Emwhitaker. A user sandbox is a subpage of the user's user page. It serves as a testing spot and page development space for the user and is not an encyclopedia article. |
| This is a user sandbox of Emwhitaker. A user sandbox is a subpage of the user's user page. It serves as a testing spot and page development space for the user and is not an encyclopedia article. |
Alcoholic hepatitis Main article: Alcoholic hepatitis
Ethanol, mostly in alcoholic beverages, is a significant cause of hepatitis. Usually alcoholic hepatitis comes after a period of increased alcohol consumption. Alcoholic hepatitis is characterized by a variable constellation of symptoms, which may include feeling unwell, enlargement of the liver, development of fluid in the abdomen ascites, and modest elevation of liver blood tests. Alcoholic hepatitis can vary from mild with only liver test elevation[2] to severe liver inflammation with development of jaundice, prolonged prothrombin time, and liver failure. Severe[3] cases are characterized by either obtundation (dulled consciousness) or the combination of elevated bilirubin levels and prolonged prothrombin time; the mortality rate in both categories is 50% within 30 days of onset.[citation needed]
Alcoholic hepatitis is distinct from cirrhosis caused by long-term alcohol consumption[4]. Alcoholic hepatitis can occur in patients with chronic alcoholic liver disease and alcoholic cirrhosis. Alcoholic hepatitis by itself does not lead to cirrhosis, but cirrhosis[5] is more common in patients with long-term alcohol consumption.[76] Patients who drink alcohol to excess are also more often than others found to have hepatitis C.[citation needed] The combination of hepatitis C and alcohol consumption accelerates the development of cirrhosis. PMID 24225087 [6] SGOT is also a problem [6]
References
- ^ . doi:10.1016/0140-6736(90)90002-M.
{{cite journal}}: Cite journal requires|journal=(help); Missing or empty|title=(help) - ^ Harabuchi, Y.; Yamanaka, N.; Kataura, A.; Imai, S.; Kinoshita, T.; Osato, T. "Epstein-Barr virus in nasal T-cell lymphomas in patients with lethal midline granuloma". The Lancet. 335 (8682): 128–130. doi:10.1016/0140-6736(90)90002-m.
- ^ Carle, A. C., & Weech-Maldonado, R. (2012). Does the Consumer Assessment of Healthcare Providers and Systems Cultural Competence Survey provide equivalent measurement across English and Spanish versions? Medical Care, 50(9 Suppl 2), S37–41. doi:10.1097/MLR.0b013e3182665189
- ^ Harabuchi, Y.; Yamanaka, N.; Kataura, A.; Imai, S.; Kinoshita, T.; Osato, T. "Epstein-Barr virus in nasal T-cell lymphomas in patients with lethal midline granuloma". The Lancet. 335 (8682): 128–130. doi:10.1016/0140-6736(90)90002-m.
- ^ Aune, D. (2012). Soft drinks, aspartame, and the risk of cancer and cardiovascular disease. The American Journal of Clinical Nutrition, 96(6), 1249–1251. doi:10.3945/ajcn.112.051417
- ^ a b Tumurbaatar B, Tikhanovich I, Li Z, Ren J, Ralston R, Kuravi S et al. (2013) Hepatitis C and Alcohol Exacerbate Liver Injury by Suppression of FOXO3. Am J Pathol ():. DOI:10.1016/j.ajpath.2013.08.013 PMID: 24225087