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Oh Uhtaek

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Template:Korean name

Oh Uhtaek
Born (1955-06-29) June 29, 1955 (age 69)
NationalitySouth Korean
Alma materKorea Institute of Science and Technology
Scientific career
FieldsDepartment of Physiology
InstitutionsProfessor, College of Pharmacy, Seoul National University
Doctoral advisorRobert D. Foreman
Oh Uhtaek
Hangul
Hanja
Revised RomanizationO Ut'aek
McCune–ReischauerO Utaek

Oh Uhtaek or Oh Woo-Taek (born June 29, 1955) is a Korean physiologist. He is also a professor at College of Pharmacy, Seoul National University. His research is largely known for Cardiac Pain and other Visceral Pain Mechanism and as well as cloning.

Education

1978 Seoul National University, College of Pharmacy, B. S.

1982 Seoul National University, College of Pharmacy, M. S.

1987 University of Oklahoma, School of Medicine, Department of Physiology, Ph. D.

Work

  • 1987 - 1988: Postdoctoral Training, Marine Biomedical Institute, University of Texas, Medical Branch at Galveston
  • 1988: Assistant, Associate, Professor, College of Pharmacy, SNU
  • 1994 - 1995: Visiting Professor, Department of Physiology, Rosalind Franklin University HSC/Chicago Medical School
  • 1997: Director, Sensory Research Center, Creative Research Initiatives, SNU
  • 1998 – 2003: Associate Editor, Neuroscience Letters, Elsevier
  • 1999 – 2000: Editor-in-chief, Archives of Pharmacal Research
  • 2000 – 2000: Secretary General, Korean Society for Brain and Neural Science
  • 2004 – 2004: Secretary General, Korean Society for Biochemistry and Molecular Biology
  • 2004 – 2004: Secretary General, Federation of Asian Pain Societies
  • 2004: Member of the Korean Academy of Science and Technology
  • 2005 – 2007: Secretary General, Organizing Committee, 19th FAOBMB Seoul Conference
  • 2007 - 2013: Treasurer, FAOBMB
  • 2008 – 2013: Chairman, WCU Dept of Molecular Medicine & Biopharmaceutical Sciences
  • 2011 - 2013: National R&D Review Board of the MEST, Board Member
  • 2011 - 2013: Biomedical Technology Development Steering Committee of MEST, Chair
  • 2013: Chairman of the Board, Institut Pasteur-Korea

Major Research Activities

  • 2012: Cho H et al., The Calcium-activated Chloride Channel Anoctamin 1 acts as a Heat Sensor in Nociceptive Neurons. Nature Neuroscience (2012)[1]
  • 2008: Yang YD et al., TMEM16A Confers Receptor Activated Calcium-dependent Chloride Conductance. Nature (2008)[2]
  • 2002: Shin J et al., Bradykinin-12-lipoxygenase-VR1 signaling pathway for inflammatory hyperalgesia. Proc Natl Acad Sci USA (2002)[3]
  • 2002: Cho H et al., Mechano-sensitive Ion Channels in Cultured Sensory Neurons of Neonatal Rats. J Neurosci (2002)[4]
  • 2000: Hwang SW et al., Direct activation of capsaicin receptors by products of lipoxygenases: Endogenous capsaicin-like substances. Proc Natl Acad Sci USA[5]
  • 1996: Oh U et al., Capsaicin Activates a Non-selective Cation Channel in Cultured Neonatal Rat Dorsal-root Ganglion Neurons. J Neurosci[6]

Awards

References

  1. ^ Oh, Utaek; Cho H; Lee, Byeongjoon; Kim, Tahnbee; Jang, Yongwoo; Back, Seung Keun; Na, Heung Sik; Harfe, Brian D; Wang, Fan; Raouf, Ramin; Wood, John N; Oh, Uhtaek (2012). "The Calcium-activated Chloride Channel Anoctamin 1 acts as a Heat Sensor in Nociceptive Neurons". Nature Neuroscience. 15 (7): 1015–21. doi:10.1038/nn.3111. PMID 22634729. {{cite journal}}: Unknown parameter |displayauthors= ignored (|display-authors= suggested) (help)
  2. ^ Oh, Utaek; Yang YD; Tak, Min Ho; Cho, Yeongyo; Shim, Won-Sik; Park, Seung Pyo; Lee, Jesun; Lee, Byeongjun; Kim, Byung-Moon; Raouf, Ramin; Shin, Young Ki; Oh, Uhtaek (2008). "TMEM16A Confers Receptor Activated Calcium-dependent Chloride Conductance". Nature. 455 (7217): 1210–5. Bibcode:2008Natur.455.1210Y. doi:10.1038/nature07313. PMID 18724360. {{cite journal}}: Unknown parameter |displayauthors= ignored (|display-authors= suggested) (help)
  3. ^ Oh, Utaek; Shin J; Jung, J.; Shin, C. Y.; Lee, S.-Y.; Kim, S. H.; Lee, M. G.; Choi, Y. H.; Kim, J.; Haber, N. A.; Reichling, D. B.; Khasar, S.; Levine, J. D.; Oh, U. (2002). "Bradykinin-12-lipoxygenase-VR1 signaling pathway for inflammatory hyperalgesia". Proc. Natl. Acad. Sci. USA. 99 (15): 10150–5. Bibcode:2002PNAS...9910150S. doi:10.1073/pnas.152002699. PMC 126639. PMID 12097645. {{cite journal}}: Unknown parameter |displayauthors= ignored (|display-authors= suggested) (help)
  4. ^ Oh, Utaek; Cho H (2002). "Mechano-sensitive Ion Channels in Cultured Sensory Neurons of Neonatal Rats". J Neurosci. {{cite journal}}: Unknown parameter |displayauthors= ignored (|display-authors= suggested) (help)
  5. ^ Oh, Utaek; Hwang SW; Kwak, J.; Lee, S.-Y.; Kang, C.-J.; Jung, J.; Cho, S.; Min, K. H.; Suh, Y.-G.; Kim, D.; Oh, U. (2000). "Direct activation of capsaicin receptors by products of lipoxygenases: Endogenous capsaicin-like substances". Proc. Natl. Acad. Sci. USA. 97 (11): 6155–60. Bibcode:2000PNAS...97.6155H. doi:10.1073/pnas.97.11.6155. PMC 18574. PMID 10823958.
  6. ^ Oh, Utaek; Oh U (1996). "Capsaicin Activates a Non-selective Cation Channel in Cultured Neonatal Rat Dorsal-root Ganglion Neurons". J Neurosci.
  7. ^ "Previous Laureates". The Ho-Am Foundation. 2019. Retrieved 19 August 2019.