Polyphagia
From Wikipedia, the free encyclopedia
In biology, "polyphagia" is a type of phagy, referring to an animal that feeds on many kinds of food.
| ICD-10 | R63.2 |
|---|---|
| ICD-9 | 783.6 |
Polyphagia is weight loss despite increased appetite (initially)[1]. It derives from the Greek words πολύς (polys) which means "very much", and φαγῶ (phago), verb for "I eat".[2]
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[edit] In medicine
In medicine, polyphagia (sometimes known as hyperphagia) is a medical sign meaning excessive hunger and abnormally large intake of solids by mouth. Disorders such as diabetes, Kleine-Levin Syndrome (a malfunction in the hypothalamus), the genetic disorders Prader-Willi Syndrome and Bardet Biedl Syndrome can cause hyperphagia (compulsive hunger).[3]
[edit] Causes
Causes of increased appetite include[4]:
- Anxiety
- Certain drugs (such as corticosteroids, cyproheptadine, and tricyclic antidepressants)
- Bulimia (most common in women 18 - 30 years old)
- Diabetes mellitus (including gestational diabetes)
- Graves' disease
- Hyperthyroidism
- Hypoglycemia
- Premenstrual syndrome
[edit] Diabetic ketoacidosis
Polyphagia usually occurs early in the course of diabetic ketoacidosis.[5] However, once insulin deficiency becomes more severe and ketoacidosis develops, appetite is suppressed.[6]
[edit] See also
[edit] References
- ^ Berthoud HR, Lenard NR, Shin AC (2011). "Food reward, hyperphagia, and obesity.". Am J Physiol Regul Integr Comp Physiol 300 (6): R1266-77. doi:10.1152/ajpregu.00028.2011. PMC 3119156. PMID 21411768. http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=21411768.
- ^ http://yourtotalhealth.ivillage.com/polyphagia.html,
- ^ OMIM::Prader-WilliOMIM::Bardet-Biedl
- ^ http://www.nlm.nih.gov/medlineplus/ency/article/003134.htm NIH Medline plus
- ^ Elliott RE, Jane JA, Wisoff JH (2011). "Surgical management of craniopharyngiomas in children: meta-analysis and comparison of transcranial and transsphenoidal approaches.". Neurosurgery 69 (3): 630–43; discussion 643. doi:10.1227/NEU.0b013e31821a872d. PMID 21499159.
- ^ Masuzaki H, Tanaka T, Ebihara K, Hosoda K, Nakao K (2009). "Hypothalamic melanocortin signaling and leptin resistance--perspective of therapeutic application for obesity-diabetes syndrome.". Peptides 30 (7): 1383–6. doi:10.1016/j.peptides.2009.04.008. PMID 19394382. http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=19394382.
[edit] External links
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