Talk:Dysmenorrhea

Level of pain

Some women say that even bad cramps are nowhere near labour pain, others say that some cramps can be worse and some gynaecologists would explain that because both sorts of pain are similar in cause and therefore the pain of each varies for woman to woman, who is right?

Contradiction

Under "Dysmenorrhea," it says dysmenorrhea "does not appear to be affected by childbearing," but under risk factors "nulliparity" is listed. Why the apparent contradiction? 72.181.235.222 23:51, 26 April 2007 (UTC)Brian Answer some women have no cramps after child bearing, but some don't.

The cited study seems to contradict the effectiveness of accupuncture for pain relief. This should probably be re-written. Jredwards 06:25, 30 April 2007 (UTC)

The sentence "although some pain is normal during menstruation, abnormal pain is not" is circular reasoning. Obviously abnormal pain is not normal. What makes this pain "abnormal" and thus distinguishes it from "normal" pain? Does the author mean excessive pain is not normal?68.101.70.151 21:21, 21 June 2007 (UTC)Britt

Yes I think most women have times in their fertile life when they have light cramps but some women have geuine dysmenhorrea.

Most women who have suffered from Dysmenorrhea had times where they didn't suffer from it - they can tell the difference. Fdskjs 19:31, 5 August 2007 (UTC)

Survey

I've just added a Norwegian study to this article. It had been incorrectly placed in the PMS article. The fantastic people at WP:WikiProject Norway provided a translation of the ref. See this discussion for more information. WhatamIdoing (talk) 19:51, 6 March 2008 (UTC)

Reimprove template

Having removed the refimprove template from this article on the basis that there were few fact tags in it and no discussion of the template here, I have been directed to previous edits which make it clear this was an error on my part. Accordingly, I have reinstated the individual fact tags in the Secondary Dysmenorrhoea section which were placed by User:Kusunose and removed by User:WhatamIdoing. It's extremely important for an editor looking to add refs to be able to see where other editors have requested them, and ideally to be able to see a rationale for those requests on the talk page.

The one I didn't reinstate related to prostaglandins, as I believe this is covered by reference 3 (Wright & Wyatt). Nmg20 (talk) 07:42, 16 May 2008 (UTC)

Kusunose did not place those ref tags; all they did was disambiguate Japan. This is why we should use diffs; then it would be clear that I was the one who added the cite requests. 66.30.20.71 (talk) 11:19, 16 May 2008 (UTC)

Unreferenced statements moved here

In this edit, I removed several unreferenced statements from the article. Here they are, if anyone would like to reference and replace them:

• Prostaglandins sensitise nociceptors to pain, so the pain threshold is lowered. Uterine contractions cause pain, which is more sensitively felt. Prostaglandin levels have been found to be much higher in females with severe menstrual pain than in females who experience mild or no menstrual pain.

• Drugs which inhibit the production of prostaglandins, such as the non-steroidal anti-inflammatory drugs (NSAIDs) naproxen, ibuprofen and mefenamic acid can provide relief for the pain associated with high prostaglandin release.

• The mechanism of action involves reducing menstrual blood volume, and suppressing ovulation. It may take up to three months of treatment to experience relief.

• Hormonal treatments such as danazol, progestational agents, GnRH agonists, and progestins may be used.

• In severe cases, interruption of uterine nerves by presacral neurectomy and division of the sacrouterine ligaments may be used when other treatment options fail.

• Approximately 24% of females who report pelvic pain are subsequently found to have endometriosis. Other causes include pelvic inflammatory disease,

• Some find that use of internally-worn menstrual products (such as tampons and menstrual cups) exacerbate dysmenorrhea.

• The mechanisms causing the pain of secondary dysmenorrhea are varied, and may or may not involve prostaglandins. Secondary dysmenorrhea is less related to the onset of bleeding in menstruation, is seen in older females, and is sometimes associated with other conditions, such as infertility.

• The most effective treatment of secondary dysmenorrhea is the identification and treatment of the underlying cause of the pain. The first line of treatment is medical; if possible, the underlying medical disorder or anatomic abnormality is corrected. Dilation of a narrow cervical os may give 3 to 6 months of relief, and allows diagnostic curettage if needed. Myomectomy, polypectomy, or dilation and curettage may be needed.

• The medical options used for relieving pain in secondary dysmenorrhea are similar to those used in primary dysmenorrhea.

• Edit: Another, removed in this edit, is here:

"While the symptoms of primary dysmenorrhea are generally limited to the time around menses, in secondary dysmenorrhea, they may extend further through the menstrual cycle."

• Edit 2: Another, removed in this edit, is here:

"The symptoms of secondary dysmenorrhea vary with the underlying cause, but are similar to those of primary dysmenorrhea."

• Edit 3: Another, removed in this edit, is here:

"An increased body temperature just before menstruation is due to prostaglandins acting on hypothalamic neurons, which are involved in body temperature regulation. The nausea and vomiting is caused by the prostaglandins acting on the chemoreceptor trigger zone."

Thanks. 66.30.20.71 (talk) 13:25, 19 June 2008 (UTC)

I have reverted one of your deletions. It was fact-tagged recently, and it is almost certainly accurate. You need to be a little slower in pulling the trigger on these. (Have you spent any time looking for citations before deleting them? While it's your 'right' to delete material, we'll get a better article sooner if you find sources instead of deleting information just because you're allowed to.) WhatamIdoing (talk) 20:17, 27 June 2008 (UTC)

Suggested citations for further improvements

I briefly looked at this article and made some changes to fix some obvious howlers in its paragraph on chiropractic care and to add some info about OCPs and whatnot, but I see that there are several more problems too. It would be nice if someone with more time could take a look at the following recent reviews on the subject, and use the information in them to improve this article.

• Harel Z (2008). "Dysmenorrhea in adolescents". Ann N Y Acad Sci. 1135: 185–95. doi:10.1196/annals.1429.007. PMID 18574224.
• Sanfilippo J, Erb T (2008). "Evaluation and management of dysmenorrhea in adolescents". Clin Obstet Gynecol. 51 (2): 257–67. doi:10.1097/GRF.0b013e31816d2307. PMID 18463457.
• French L (2008). "Dysmenorrhea in adolescents: diagnosis and treatment". Paediatr Drugs. 10 (1): 1–7. PMID 18162003.
• Proctor ML, Murphy PA (2001). "Herbal and dietary therapies for primary and secondary dysmenorrhoea". Cochrane Database Syst Rev (3): CD002124. doi:10.1002/14651858.CD002124. PMID 11687013.
• Marjoribanks J, Proctor ML, Farquhar C (2003). "Nonsteroidal anti-inflammatory drugs for primary dysmenorrhoea". Cochrane Database Syst Rev (4): CD001751. doi:10.1002/14651858.CD001751. PMID 14583938.{{cite journal}}: CS1 maint: multiple names: authors list (link)
• Proctor ML, Latthe PM, Farquhar CM, Khan KS, Johnson NP (2005). "Surgical interruption of pelvic nerve pathways for primary and secondary dysmenorrhoea". Cochrane Database Syst Rev (4): CD001896. doi:10.1002/14651858.CD001896.pub2. PMID 16235288.{{cite journal}}: CS1 maint: multiple names: authors list (link)

Eubulides (talk) 08:09, 6 July 2008 (UTC)

Added "Explanation" section.

Not too long ago, I was very, very upset when I came here and saw that this article had absolutely no explanation as to what causes menstrual cramps, so I took matters into my own hands and added it myself. Morons. Pastel kitten (talk) 20:36, 5 September 2008 (UTC)