Jane Stewart (scientist)

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Jane Stewart
Alma mater Queen's University, University of London
Scientific career
Fields Neuroscience
Institutions Ayerst Pharmaceuticals, Concordia University

Jane Stewart, OC is a Canadian neuroscientist who has been active in the fields of psychology, psychiatry, and psychopharmacology.

Career[edit]

Stewart received her PhD in 1959 from the University of London, England.[1] She then started working for Ayerst Pharmaceuticals in Montreal and subsequently joined Concordia University in 1962,[2] where she served as chair of the Department of Psychology (1969–1974) and director of the Center for Studies in Behavioral Neurobiology (1990–1997).[1] She served on many grant review committees and on the editorial boards of 11 peer-reviewed scientific journals.[1]

Research[edit]

Stewart has made seminal contributions to different areas of research, such as conditioned drug effects,[3][4] the motivational effects of drugs,[5] circadian rhythms,[6] antidepressant and antipsychotic drug action,[7][8] and sexual behavior.[9][10]

Honors[edit]

Stewart obtained an honorary degree from Queen's University and is a Fellow of the American Association for the Advancement of Science, the American Psychological Association, the Canadian Psychological Association, and the Royal Society of Canada, Academy of Sciences.[1] She also received the highest civilian honor in her country, being appointed Officer in the Order of Canada in 2007.[1] A special issue of the journal Biological Psychiatry was dedicated to her on the occasion of her retirement in 2008.[1]

Significant papers[edit]

References[edit]

  1. ^ a b c d e f de Wit H, Shaham Y (May 2009). "Incentive motivation, conditioning, stress, and neuropsychiatric disorders: A tribute to Jane Stewart". Biological Psychiatry. 65 (10): 827–8. doi:10.1016/j.biopsych.2008.12.012. PMC 2716031Freely accessible. PMID 19398047. Retrieved 2009-05-06. 
  2. ^ "Jane Stewart". www.concordia.ca. Retrieved 2016-03-09. 
  3. ^ Eikelboom R, Stewart J (September 1982). "Conditioning of drug-induced physiological responses". Psychological Review. 89 (5): 507–28. doi:10.1037/0033-295X.89.5.507. PMID 7178331. 
  4. ^ Stewart J (June 1992). "Neurobiology of conditioning to drugs of abuse". Annals of the New York Academy of Sciences. 654: 335–46. doi:10.1111/j.1749-6632.1992.tb25979.x. PMID 1321575. 
  5. ^ Stewart J, de Wit H, Eikelboom R (April 1984). "Role of unconditioned and conditioned drug effects in the self-administration of opiates and stimulants". Psychological Review. 91 (2): 251–68. doi:10.1037/0033-295X.91.2.251. PMID 6571424. 
  6. ^ Amir S, Stewart J (February 1996). "Resetting of the circadian clock by a conditioned stimulus". Nature. 379 (6565): 542–5. doi:10.1038/379542a0. PMID 8596633. 
  7. ^ Stewart J, Rajabi H (August 1996). "Initial increases in extracellular dopamine in the ventral tegmental area provide a mechanism for the development of desipramine-induced sensitization within the midbrain dopamine system". Synapse. 23 (4): 258–64. doi:10.1002/(SICI)1098-2396(199608)23:4<258::AID-SYN3>3.0.CO;2-6. PMID 8855510. 
  8. ^ Samaha AN, Seeman P, Stewart J, Rajabi H, Kapur S (March 2007). ""Breakthrough" dopamine supersensitivity during ongoing antipsychotic treatment leads to treatment failure over time". Journal of Neuroscience. 27 (11): 2979–86. doi:10.1523/JNEUROSCI.5416-06.2007. PMID 17360921. Retrieved 2009-05-06. 
  9. ^ Mitchell JB, Stewart J (March 1990). "Facilitation of sexual behaviors in the male rat associated with intra-VTA injections of opiates". Pharmacology Biochemistry and Behavior. 35 (3): 643–50. doi:10.1016/0091-3057(90)90302-X. PMID 1971113. 
  10. ^ Mitchell JB, Stewart J (February 1990). "Facilitation of sexual behaviors in the male rat in the presence of stimuli previously paired with systemic injections of morphine". Pharmacology Biochemistry and Behavior. 35 (2): 367–72. doi:10.1016/0091-3057(90)90171-D. PMID 2320644. Retrieved 2009-05-06. 
  11. ^ a b c d e Web of Science, accessed May 6, 2009