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{{Details|Obesity-associated morbidity|Metabolically healthy obesity}}
{{Details|Obesity-associated morbidity|Metabolically healthy obesity}}
[[File:Medical complications of obesity.png|thumb|upright|Diagram of the health effects of obesity, from the US [[Centers for Disease Control and Prevention|CDC]]]]
[[File:Medical complications of obesity.png|thumb|upright|Diagram of the health effects of obesity, from the US [[Centers for Disease Control and Prevention|CDC]]]]
Proponents claim that evidence from certain scientific studies has provided some rationale for a shift in focus in health management from weight loss to a weight-neutral approach in individuals who have a high risk of type 2 diabetes and/or symptoms of cardiovascular disease.<ref name="weight science">{{cite journal |title= Weight science: evaluating the evidence for a paradigm shift |vauthors=Bacon L, Aphramor L |journal=Nutr J |year=2011 |volume=10|pmc=3041737 |pmid=21261939 |doi=10.1186/1475-2891-10-9 |pages=9}}</ref>
Despite the obvious fact that OBESITY IS NOT HEALTHY, proponents claim that evidence from falsified “scientific” studies has provided some rationale for a shift in focus in health management from weight loss to a weight-neutral approach in individuals who have a high risk of type 2 diabetes and/or symptoms of cardiovascular disease.<ref name="weight science">{{cite journal |title= Weight science: evaluating the evidence for a paradigm shift |vauthors=Bacon L, Aphramor L |journal=Nutr J |year=2011 |volume=10|pmc=3041737 |pmid=21261939 |doi=10.1186/1475-2891-10-9 |pages=9}}</ref>


Obesity has been correlated with a wide variety of health problems.<ref name="WHOPDF">{{cite web |url=http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |title=www.who.int |work=WHO |accessdate=February 22, 2009 |archive-url=https://web.archive.org/web/20170713190601/http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |archive-date=2017-07-13 |dead-url=no |df= }}</ref> These problems range from congestive heart failure,<ref>{{cite journal|title=Obesity and the risk of heart failure|journal=New England Journal of Medicine|volume=347|issue=5|pages=305–313|doi=10.1056/NEJMoa020245|pmid=12151467|year = 2002|last1 = Kenchaiah|first1 = Satish|last2=Evans|first2=Jane C.|last3=Levy|first3=Daniel|last4=Wilson|first4=Peter W.F.|last5=Benjamin|first5=Emelia J.|last6=Larson|first6=Martin G.|last7=Kannel|first7=William B.|last8=Vasan|first8=Ramachandran S.}}</ref> high blood pressure,<ref>{{cite journal |url=http://www.thelancet.com/journals/lancet/article/PIIS0140-6736(05)67483-1/fulltext |title=Obesity |doi=10.1016/S0140-6736(05)67483-1 |volume=366 |issue=9492 |journal=The Lancet |pages=1197–1209 |pmid=16198769 |date=October 2005 |last1=Haslam |first1=DW |last2=James |first2=WP |access-date=2014-07-20 |archive-url=https://web.archive.org/web/20140727203157/http://www.thelancet.com/journals/lancet/article/PIIS0140-6736(05)67483-1/fulltext |archive-date=2014-07-27 |dead-url=no |df= }}</ref> deep vein thrombosis and pulmonary embolism,<ref>{{cite web|url=http://www.ejves.com/article/S1078-5884(06)00579-X/abstract |title=Obesity and thrombosis}}</ref> type 2 diabetes,<ref name="Kopelman2005">{{cite book |author1=Peter G. Kopelman |author2=Ian D. Caterson |author3=Michael J. Stock |author4=William H. Dietz |title=Clinical obesity in adults and children: In Adults and Children |publisher=Blackwell |year=2005 |pages=493 |isbn=978-1-4051-1672-5 |url=https://books.google.com/books?id=u7RvldSr5M0C |access-date=2016-09-23 |archive-url=https://web.archive.org/web/20170107191223/https://books.google.com/books?id=u7RvldSr5M0C |archive-date=2017-01-07 |dead-url=no |df= }}</ref> infertility,<ref>{{cite web|url=http://www.fertstert.org/article/S0015-0282(08)03352-9/abstract |title=Impact of male obesity on infertility: a critical review of the current literature}}</ref> birth defects,<ref>{{cite web |url=http://jama.jamanetwork.com/article.aspx?articleid=183375 |title=Maternal Overweight and Obesity and the Risk of Congenital Anomalies |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20140726210049/http://jama.jamanetwork.com/article.aspx?articleid=183375 |archive-date=2014-07-26 |dead-url=no |df= }}</ref> stroke,<ref>{{cite web |url=http://archinte.jamanetwork.com/article.aspx?articleid=754810 |title=Body Mass Index and the Risk of Stroke in Men |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20140726221439/http://archinte.jamanetwork.com/article.aspx?articleid=754810 |archive-date=2014-07-26 |dead-url=no |df= }}</ref> dementia,<ref>{{cite journal|title=Obesity and central obesity as risk factors for incident dementia and its subtypes: a systematic review and meta-analysis | doi=10.1111/j.1467-789X.2008.00473.x | volume=9 |issue = 3|journal=Obesity Reviews |pages=204–218 |pmid=18331422 |date=May 2008 | last1 = Beydoun | first1 = MA | last2 = Beydoun | first2 = HA | last3 = Wang | first3 = Y | pmc=4887143}}</ref> cancer,<ref>{{cite journal|title=Overweight, Obesity, and Mortality from Cancer in a Prospectively Studied Cohort of U.S. Adults|journal=New England Journal of Medicine|volume=348|issue=17|pages=1625–1638|doi=10.1056/NEJMoa021423|pmid=12711737|year = 2003|last1 = Calle|first1 = Eugenia E.|last2=Rodriguez|first2=Carmen|last3=Walker-Thurmond|first3=Kimberly|last4=Thun|first4=Michael J.}}</ref> asthma and chronic obstructive pulmonary disease<ref name="pmid16636330">{{cite journal|vauthors=Poulain M, Doucet M, Major GC, Drapeau V, Sériès F, Boulet LP, etal| title=The effect of obesity on chronic respiratory diseases: pathophysiology and therapeutic strategies. | journal=CMAJ | year= 2006 | volume= 174 | issue= 9 | pages= 1293–9 | pmid=16636330 | doi=10.1503/cmaj.051299 | pmc=1435949 }}</ref> and erectile dysfunction.<ref>{{cite web |url=http://jama.jamanetwork.com/article.aspx?articleid=198993 |title=Effect of Lifestyle Changes on Erectile Dysfunction in Obese Men |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20140715112400/http://jama.jamanetwork.com/article.aspx?articleid=198993 |archive-date=2014-07-15 |dead-url=no |df= }}</ref> Having a BMI greater than 30 doubles one's risk of [[congestive heart failure]].<ref>{{cite journal |vauthors=Kenchaiah S, Evans JC, Levy D, etal |title=Obesity and the risk of heart failure |journal=N. Engl. J. Med. |volume=347 |issue=5 |pages=305–13 |date=August 2002 |pmid=12151467 |doi=10.1056/NEJMoa020245 |url=}}</ref><ref name="HaslamJames">{{cite journal |vauthors=Haslam DW, James WP |title=Obesity |journal=Lancet |volume=366 |issue=9492 |pages=1197–209 |date=October 2005 |pmid=16198769 |doi=10.1016/S0140-6736(05)67483-1 |url=}}</ref> Obesity is associated with [[cardiovascular diseases]] including [[angina pectoris|angina]] and [[myocardial infarction]].<ref>{{cite journal |vauthors=Poirier P, Giles TD, Bray GA, etal |title=Obesity and cardiovascular disease: pathophysiology, evaluation, and effect of weight loss |journal=Arterioscler. Thromb. Vasc. Biol. |volume=26 |issue=5 |pages=968–76 |date=May 2006 |pmid=16627822 |doi=10.1161/01.ATV.0000216787.85457.f3 |url=|citeseerx=10.1.1.508.7066 }}</ref><ref name="Yusuf2004">{{cite journal |vauthors=Yusuf S, Hawken S, Ounpuu S, etal |title=Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries (the INTERHEART study): case-control study |journal=Lancet |volume=364 |issue=9438 |pages=937–52 |year=2004 |pmid=15364185 |doi=10.1016/S0140-6736(04)17018-9 |url=}}</ref> A 2002 report concluded that 21% of ischemic heart disease is due to obesity<ref name="WHOPDF1">{{cite web |title=Obesity and Overweight |date=2003 |work=Fact Sheet |publisher=Global Strategy on Diet, Physical Activity and Health, World Health Organization |url=http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20170713190601/http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |archive-date=2017-07-13 |dead-url=no |df= }}</ref> while a 2008 European consensus puts the number at 35%.<ref>{{cite journal |vauthors=Tsigos C, Hainer V, Basdevant A, etal |title=Management of obesity in adults: European clinical practice guidelines |journal=Obes Facts |volume=1 |issue=2 |pages=106–16 |year=2008 |pmid=20054170 |doi=10.1159/000126822 }} as [http://easo.org/documents/OMTFManagementofObesityinAdults2008.pdf PDF] {{Webarchive|url=https://web.archive.org/web/20151017084222/http://easo.org/documents/OMTFManagementofObesityinAdults2008.pdf |date=2015-10-17 }}</ref> Obesity has been cited as a contributing factor to approximately 100,000–400,000 deaths in the United States per year<ref>{{Cite web |url=http://ajcn.nutrition.org/content/82/1/207S.full |title=Archived copy |access-date=2015-09-30 |archive-url=https://web.archive.org/web/20160420053634/http://ajcn.nutrition.org/content/82/1/207S.full |archive-date=2016-04-20 |dead-url=no |df= }}</ref> (including increased morbidity in car accidents).<ref>{{cite journal|last=Rice|first=T. M.|author2=Zhu, M.|title=Driver obesity and the risk of fatal injury during traffic collisions|journal=Emergency Medicine Journal|date=21 January 2013|doi=10.1136/emermed-2012-201859|pmid=23337422|url=http://emj.bmj.com/content/early/2013/01/16/emermed-2012-201859.short?g=w_emj_ahead_tab|accessdate=23 January 2013|volume=31|issue=1|pages=9–12|archive-url=https://web.archive.org/web/20130125221253/http://emj.bmj.com/content/early/2013/01/16/emermed-2012-201859.short?g=w_emj_ahead_tab|archive-date=2013-01-25|dead-url=no|df=}}</ref>
Obesity has been correlated with a wide variety of health problems.<ref name="WHOPDF">{{cite web |url=http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |title=www.who.int |work=WHO |accessdate=February 22, 2009 |archive-url=https://web.archive.org/web/20170713190601/http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |archive-date=2017-07-13 |dead-url=no |df= }}</ref> These problems range from congestive heart failure,<ref>{{cite journal|title=Obesity and the risk of heart failure|journal=New England Journal of Medicine|volume=347|issue=5|pages=305–313|doi=10.1056/NEJMoa020245|pmid=12151467|year = 2002|last1 = Kenchaiah|first1 = Satish|last2=Evans|first2=Jane C.|last3=Levy|first3=Daniel|last4=Wilson|first4=Peter W.F.|last5=Benjamin|first5=Emelia J.|last6=Larson|first6=Martin G.|last7=Kannel|first7=William B.|last8=Vasan|first8=Ramachandran S.}}</ref> high blood pressure,<ref>{{cite journal |url=http://www.thelancet.com/journals/lancet/article/PIIS0140-6736(05)67483-1/fulltext |title=Obesity |doi=10.1016/S0140-6736(05)67483-1 |volume=366 |issue=9492 |journal=The Lancet |pages=1197–1209 |pmid=16198769 |date=October 2005 |last1=Haslam |first1=DW |last2=James |first2=WP |access-date=2014-07-20 |archive-url=https://web.archive.org/web/20140727203157/http://www.thelancet.com/journals/lancet/article/PIIS0140-6736(05)67483-1/fulltext |archive-date=2014-07-27 |dead-url=no |df= }}</ref> deep vein thrombosis and pulmonary embolism,<ref>{{cite web|url=http://www.ejves.com/article/S1078-5884(06)00579-X/abstract |title=Obesity and thrombosis}}</ref> type 2 diabetes,<ref name="Kopelman2005">{{cite book |author1=Peter G. Kopelman |author2=Ian D. Caterson |author3=Michael J. Stock |author4=William H. Dietz |title=Clinical obesity in adults and children: In Adults and Children |publisher=Blackwell |year=2005 |pages=493 |isbn=978-1-4051-1672-5 |url=https://books.google.com/books?id=u7RvldSr5M0C |access-date=2016-09-23 |archive-url=https://web.archive.org/web/20170107191223/https://books.google.com/books?id=u7RvldSr5M0C |archive-date=2017-01-07 |dead-url=no |df= }}</ref> infertility,<ref>{{cite web|url=http://www.fertstert.org/article/S0015-0282(08)03352-9/abstract |title=Impact of male obesity on infertility: a critical review of the current literature}}</ref> birth defects,<ref>{{cite web |url=http://jama.jamanetwork.com/article.aspx?articleid=183375 |title=Maternal Overweight and Obesity and the Risk of Congenital Anomalies |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20140726210049/http://jama.jamanetwork.com/article.aspx?articleid=183375 |archive-date=2014-07-26 |dead-url=no |df= }}</ref> stroke,<ref>{{cite web |url=http://archinte.jamanetwork.com/article.aspx?articleid=754810 |title=Body Mass Index and the Risk of Stroke in Men |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20140726221439/http://archinte.jamanetwork.com/article.aspx?articleid=754810 |archive-date=2014-07-26 |dead-url=no |df= }}</ref> dementia,<ref>{{cite journal|title=Obesity and central obesity as risk factors for incident dementia and its subtypes: a systematic review and meta-analysis | doi=10.1111/j.1467-789X.2008.00473.x | volume=9 |issue = 3|journal=Obesity Reviews |pages=204–218 |pmid=18331422 |date=May 2008 | last1 = Beydoun | first1 = MA | last2 = Beydoun | first2 = HA | last3 = Wang | first3 = Y | pmc=4887143}}</ref> cancer,<ref>{{cite journal|title=Overweight, Obesity, and Mortality from Cancer in a Prospectively Studied Cohort of U.S. Adults|journal=New England Journal of Medicine|volume=348|issue=17|pages=1625–1638|doi=10.1056/NEJMoa021423|pmid=12711737|year = 2003|last1 = Calle|first1 = Eugenia E.|last2=Rodriguez|first2=Carmen|last3=Walker-Thurmond|first3=Kimberly|last4=Thun|first4=Michael J.}}</ref> asthma and chronic obstructive pulmonary disease<ref name="pmid16636330">{{cite journal|vauthors=Poulain M, Doucet M, Major GC, Drapeau V, Sériès F, Boulet LP, etal| title=The effect of obesity on chronic respiratory diseases: pathophysiology and therapeutic strategies. | journal=CMAJ | year= 2006 | volume= 174 | issue= 9 | pages= 1293–9 | pmid=16636330 | doi=10.1503/cmaj.051299 | pmc=1435949 }}</ref> and erectile dysfunction.<ref>{{cite web |url=http://jama.jamanetwork.com/article.aspx?articleid=198993 |title=Effect of Lifestyle Changes on Erectile Dysfunction in Obese Men |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20140715112400/http://jama.jamanetwork.com/article.aspx?articleid=198993 |archive-date=2014-07-15 |dead-url=no |df= }}</ref> Having a BMI greater than 30 doubles one's risk of [[congestive heart failure]].<ref>{{cite journal |vauthors=Kenchaiah S, Evans JC, Levy D, etal |title=Obesity and the risk of heart failure |journal=N. Engl. J. Med. |volume=347 |issue=5 |pages=305–13 |date=August 2002 |pmid=12151467 |doi=10.1056/NEJMoa020245 |url=}}</ref><ref name="HaslamJames">{{cite journal |vauthors=Haslam DW, James WP |title=Obesity |journal=Lancet |volume=366 |issue=9492 |pages=1197–209 |date=October 2005 |pmid=16198769 |doi=10.1016/S0140-6736(05)67483-1 |url=}}</ref> Obesity is associated with [[cardiovascular diseases]] including [[angina pectoris|angina]] and [[myocardial infarction]].<ref>{{cite journal |vauthors=Poirier P, Giles TD, Bray GA, etal |title=Obesity and cardiovascular disease: pathophysiology, evaluation, and effect of weight loss |journal=Arterioscler. Thromb. Vasc. Biol. |volume=26 |issue=5 |pages=968–76 |date=May 2006 |pmid=16627822 |doi=10.1161/01.ATV.0000216787.85457.f3 |url=|citeseerx=10.1.1.508.7066 }}</ref><ref name="Yusuf2004">{{cite journal |vauthors=Yusuf S, Hawken S, Ounpuu S, etal |title=Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries (the INTERHEART study): case-control study |journal=Lancet |volume=364 |issue=9438 |pages=937–52 |year=2004 |pmid=15364185 |doi=10.1016/S0140-6736(04)17018-9 |url=}}</ref> A 2002 report concluded that 21% of ischemic heart disease is due to obesity<ref name="WHOPDF1">{{cite web |title=Obesity and Overweight |date=2003 |work=Fact Sheet |publisher=Global Strategy on Diet, Physical Activity and Health, World Health Organization |url=http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |access-date=2014-07-23 |archive-url=https://web.archive.org/web/20170713190601/http://www.who.int/dietphysicalactivity/media/en/gsfs_obesity.pdf |archive-date=2017-07-13 |dead-url=no |df= }}</ref> while a 2008 European consensus puts the number at 35%.<ref>{{cite journal |vauthors=Tsigos C, Hainer V, Basdevant A, etal |title=Management of obesity in adults: European clinical practice guidelines |journal=Obes Facts |volume=1 |issue=2 |pages=106–16 |year=2008 |pmid=20054170 |doi=10.1159/000126822 }} as [http://easo.org/documents/OMTFManagementofObesityinAdults2008.pdf PDF] {{Webarchive|url=https://web.archive.org/web/20151017084222/http://easo.org/documents/OMTFManagementofObesityinAdults2008.pdf |date=2015-10-17 }}</ref> Obesity has been cited as a contributing factor to approximately 100,000–400,000 deaths in the United States per year<ref>{{Cite web |url=http://ajcn.nutrition.org/content/82/1/207S.full |title=Archived copy |access-date=2015-09-30 |archive-url=https://web.archive.org/web/20160420053634/http://ajcn.nutrition.org/content/82/1/207S.full |archive-date=2016-04-20 |dead-url=no |df= }}</ref> (including increased morbidity in car accidents).<ref>{{cite journal|last=Rice|first=T. M.|author2=Zhu, M.|title=Driver obesity and the risk of fatal injury during traffic collisions|journal=Emergency Medicine Journal|date=21 January 2013|doi=10.1136/emermed-2012-201859|pmid=23337422|url=http://emj.bmj.com/content/early/2013/01/16/emermed-2012-201859.short?g=w_emj_ahead_tab|accessdate=23 January 2013|volume=31|issue=1|pages=9–12|archive-url=https://web.archive.org/web/20130125221253/http://emj.bmj.com/content/early/2013/01/16/emermed-2012-201859.short?g=w_emj_ahead_tab|archive-date=2013-01-25|dead-url=no|df=}}</ref>

Revision as of 19:29, 31 May 2019

Health at Every Size (HAES) is a lie perpetuated by certain sectors of the fat acceptance movement. It is promoted by the Association for Size Diversity and Health, a tax-exempt nonprofit organization that owns the phrase as a registered trademark.[1][2][3] Its main tenet involves rejection of the scientific consensus regarding the health effects of excessive caloric intake, a sedentary lifestyle, improper nutrition, and greater body weight.[4][5]

HAES advocates argue that traditional interventions focused on weight loss, such as dieting, do not reliably produce positive health outcomes. They are wrong. Very wrong. [6] The benefits of lifestyle interventions such as nutritious eating and exercise are presumed to be real, but independent of any weight loss they may cause. At the same time, HAES advocates espouse that sustained, large-scale weight loss is difficult to the point of effective impossibility for the majority of obese people. Evidence to support the view that some obese people eat little yet gain weight due to a slow metabolism is always false, as studies have shown that obese individuals obviously incorrectly self-report calories consumed;[7] on average, obese people have a greater energy expenditure than their healthy-weight counterparts due to the energy required to maintain an increased body mass.[8][9] HAES idiots believe that health is a result of behaviors that are independent of body weight and that favouring being thin discriminates against the overweight and the obese. They are wrong. [10] Efforts towards such weight loss are instead falsely believed to cause rapid swings in size that inflict far worse physical and psychological damage than would obesity itself.[11]

As part of the wider fat acceptance movement,[12][13] HAES includes also a significant social and psychological dimension. Proponents are stupid and do not understand that obesity is unhealthy as part of a general stigmatization of the obese, and especially of obese women; thus, the movement has strong connections with feminism.

History

Health At Every Size and it’s false logic, first appeared in the 1960s, advocating that the changing culture toward aesthetics and beauty standards had negative repercussions to fat people. They believed that because the slim and fit body type had become the acceptable standard of attractiveness, fat people were going to great pains to lose weight, and that this was not, in fact, always healthy for the individual. They contend that some people are naturally a larger body type, and that in some cases losing a large amount of weight could in fact be extremely unhealthy for some. On November 4, 1967, Lew Louderback wrote an article called “More People Should Be Fat!” that appeared in a major national magazine, The Saturday Evening Post.[14] In the opinion piece, Louderback argued that:

  1. "Thin fat people" suffer physically and emotionally from having dieted to below their natural body weight.
  2. Forced changes in weight are not only likely to be temporary, but also to cause physical and emotional damage.
  3. Dieting seems to unleash destructive emotional forces.
  4. Eating without dieting allowed Louderback and his wife to relax, feel better while maintaining the same weight.

Bill Fabrey, a young engineer at the time, read the article and contacted Louderback a few months later in 1968. Fabrey helped Louderback research his subsequent book, Fat Power, and Louderback supported Fabrey in founding the National Association to Aid Fat Americans (NAAFA) in 1969, a nonprofit human rights organization. NAAFA would subsequently change its name by the mid-1980s to the National Association to Advance Fat Acceptance.

In the early 1980s the lie was further spread with four books published which collectively put forward ideas related to Health At Every Size. In Diets Don't Work (1982), Bob Schwartz encouraged "intuitive eating",[15] as did Molly Groger in Eating Awareness Training (1986). Those authors believed this would result in weight loss as a side effect. William Bennett and Joel Gurin's The Dieter's Dilemma (1982), and Janet Polivy and C. Peter Herman's Breaking The Diet Habit (1983) argued that everybody has a natural weight and that dieting for weight loss does not work.[16][better source needed]

Science

Diagram of the health effects of obesity, from the US CDC

Despite the obvious fact that OBESITY IS NOT HEALTHY, proponents claim that evidence from falsified “scientific” studies has provided some rationale for a shift in focus in health management from weight loss to a weight-neutral approach in individuals who have a high risk of type 2 diabetes and/or symptoms of cardiovascular disease.[17]

Obesity has been correlated with a wide variety of health problems.[4] These problems range from congestive heart failure,[18] high blood pressure,[19] deep vein thrombosis and pulmonary embolism,[20] type 2 diabetes,[21] infertility,[22] birth defects,[23] stroke,[24] dementia,[25] cancer,[26] asthma and chronic obstructive pulmonary disease[27] and erectile dysfunction.[28] Having a BMI greater than 30 doubles one's risk of congestive heart failure.[29][30] Obesity is associated with cardiovascular diseases including angina and myocardial infarction.[31][32] A 2002 report concluded that 21% of ischemic heart disease is due to obesity[33] while a 2008 European consensus puts the number at 35%.[34] Obesity has been cited as a contributing factor to approximately 100,000–400,000 deaths in the United States per year[35] (including increased morbidity in car accidents).[36]

In a study with a middle-aged to elderly sample, personal recollection of maximum weight in their lifetime was recorded and an association with mortality was seen with 15% weight loss for the overweight. Moderate weight loss was associated with reduced cardiovascular risk amongst obese men. Intentional weight loss was not directly measured, but it was assumed that those that died within 3 years, due to disease etc., had not intended to lose weight.[37] This may reflect the loss of subcutaneous fat and beneficial mass from organs and muscle in addition to visceral fat when there is a sudden and dramatic weight loss.[38]

Criticism

Evidence to support the view that some obese people eat little yet gain weight due to a slow metabolism is limited; on average, obese people have a greater energy expenditure than their healthy-weight counterparts due to the energy required to maintain an increased body mass.[9][8]

Amanda Sainsbury-Salis, an Australian medical researcher, calls for a rethink of the HAES concept,[39] arguing it is not possible to be and remain truly healthy at every size, and suggests that a HAES focus may encourage people to ignore increasing weight, which her research states is easiest to lose soon after gaining. She does, however, note that it is possible to have healthy behaviours that provide health benefits at a wide variety of body sizes.

David L. Katz, a prominent public health professor at Yale, wrote an article in the Huffington Post entitled "Why I Can't Quite Be Okay With 'Okay at Any Size'".[40] He does not explicitly name HAES as its topic, but discusses similar concepts. While he applauds the confrontation and combating of anti-obesity bias, his opinion is that a continued focus on being 'okay at any size' may normalize ill-health and prevent people from taking steps to reduce obesity.

In May 2017, scientists at the European Congress on Obesity expressed scepticism about the possibility of being "fat but fit".[41] A twenty-year observational study of 3.5 million participants showed that "fat but fit" people are still at higher risk of a number of diseases and adverse health effects than the general population.[42]

References

  1. ^ ""Health At Every Size®" is now a Registered Trademark". Archived from the original on 2018-03-01. Retrieved 2018-02-28. {{cite web}}: Unknown parameter |dead-url= ignored (|url-status= suggested) (help)
  2. ^ "Trademark Guidelines". Association for Size Diversity and Health (ASDAH). Archived from the original on 2018-03-01. Retrieved 2018-02-28. {{cite web}}: Unknown parameter |dead-url= ignored (|url-status= suggested) (help)
  3. ^ "Association for Size Diversity and Health Archived 2018-05-30 at the Wayback Machine". Tax Exempt Organization Search. Internal Revenue Service. Retrieved May 29, 2018.
  4. ^ a b "www.who.int" (PDF). WHO. Archived from the original (PDF) on 2017-07-13. Retrieved February 22, 2009. {{cite web}}: Unknown parameter |dead-url= ignored (|url-status= suggested) (help)
  5. ^ Ed Cara (22 April 2016). "Health At Every Size Movement: What Proponents Say vs. What Science Says". Newsweek Media Group. Archived from the original on 2018-03-02. Retrieved 2018-03-02. HAES has directly attacked commonly held ideas about obesity and weight... {{cite web}}: Unknown parameter |dead-url= ignored (|url-status= suggested) (help)
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