Hyperventilation: Difference between revisions
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==Treatment== |
==Treatment== |
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{{Unreferenced section|date=November 2008}} |
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The first step that should be taken is to treat the underlying cause of the hyperventilation. The patient should be encouraged to control his/her breathing. If this cannot be achieved, supplemental oxygen may be given to reduce tissue hypoxia. |
The first step that should be taken is to treat the underlying cause of the hyperventilation. The patient should be encouraged to control his/her breathing. If this cannot be achieved, supplemental oxygen may be given to reduce tissue hypoxia. For possible [[Behavior_therapy|behavior therapy]] see [[Hyperventilation_syndrome#Treatment|Treatment]] in [[Hyperventilation_syndrome|Hyperventilation syndrome]]. Drug management is sometimes necessary. Parenteral drugs may have to be administered to reduce the patients anxiety and to slow the rate of breathing. [[Diazepam]] or [[midazolam]] are sometimes used. |
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Have the patient lie in semi-erect position. If patient is conscious, ask him/her to rebreath into paper bags to increase inspired co2 and to overcome alkalization. Note, however, that some physicians do not advise the paper bag rebreathing method (or limiting its use to one or two minutes) due to the possibility of inhaling too much carbon dioxide and decreasing inspired oxygen to a hypoxic patient. |
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If the patient is unconscious, maintain proper airway until he/she regains consciousness. This condition is a self limiting one and eventually the patient will settle. |
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For possible [[Behavior_therapy|behavior therapy]] see [[Hyperventilation_syndrome#Treatment|Treatment]] in [[Hyperventilation_syndrome|Hyperventilation syndrome]]. |
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Drug management is sometimes necessary. Parenteral drugs may have to be administered to reduce the patients anxiety and to slow the rate of breathing. [[Diazepam]] or [[midazolam]] are sometimes used. |
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==References== |
==References== |
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Revision as of 07:20, 18 November 2009
| Hyperventilation |
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In medicine, hyperventilation (or overbreathing) is the state of breathing faster and/or deeper than necessary, bringing about lightheadedness and other undesirable symptoms often associated with panic attacks. Hyperventilation can also be a response to metabolic acidosis, a condition that causes acidic blood pH levels.
Counterintuitively, such side effects are not precipitated by the sufferer's lack of oxygen or air. Rather, the hyperventilation itself reduces the carbon dioxide concentration of the blood to below its normal level, thereby raising the blood's pH value (making it more alkaline), initiating constriction of the blood vessels which supply the brain, and preventing the transport of certain electrolytes necessary for the function of the nervous system.[1]
Template:Lung size/activity Hyperventilation can, but does not necessarily always cause symptoms such as numbness or tingling in the hands, feet and lips, lightheadedness, dizziness, headache, chest pain, slurred speech and sometimes fainting, particularly when accompanied by the Valsalva maneuver. Sometimes hyperventilation is induced for these same effects.
Causes
Stress or anxiety commonly are causes of hyperventilation; this is known as hyperventilation syndrome. Hyperventilation can also be brought about voluntarily, by taking many deep breaths. Hyperventilation can also occur as a consequence of various lung diseases, head injury, or stroke (central neurogenic hyperventilation, apneustic respirations, ataxic respiration, Cheyne-Stokes respirations or Biot's respiration). Lastly, in the case of metabolic acidosis, the body uses hyperventilation as a compensatory mechanism to decrease acidity of the blood. In the setting of Diabetic Ketoacidosis, this is known as Kussmaul breathing - characterized by long, deep breaths.
Hyperventilation can also occur when someone exercises over his/her VO2 max, when he/she can't transform oxygen into energy beyond a certain level but hyperventilates in an effort to do so.
Hyperventilation is not the same as hyperpnoea. In hyperpnoea, increased ventilation is appropriate for a metabolic acidotic state, this is also known as respiratory compensation. Whereas in hyperventilation, increased ventilation is inappropriate for the metabolic state of blood plasma.
Mechanism
In normal breathing, both the depth and frequency of breaths are varied by the neural (or, nervous) system, primarily in order to maintain normal amounts of carbon dioxide but also to supply appropriate levels of oxygen to the body's tissues. This is mainly achieved by measuring the carbon dioxide content of the blood; normally, a high carbon dioxide concentration signals a low oxygen concentration, as we breathe in oxygen and breathe out carbon dioxide at the same time, and the body's cells use oxygen to burn fuel molecules, making carbon dioxide as a by-product.
The gases in the alveoli of the lungs are nearly in equilibrium with the gases in the blood. Normally, less than 10% of the gas in the alveoli is replaced with each breath taken. Deeper or quicker breaths exchange more of the alveolar gas with ambient air and have the net effect of expelling more carbon dioxide from the body, since the carbon dioxide concentration in normal air is very low.
The resulting low concentration of carbon dioxide in the blood is known as hypocapnia. Since carbon dioxide is carried as carbonic acid in the blood, hypocapnia results in the blood becoming alkaline, i.e. the blood pH value rises. This is known as a respiratory alkalosis.
Hypocapnia can cause a reduction in the respiratory drive.
If carbon dioxide levels are high, the body assumes that oxygen levels are low, and accordingly, the brain's blood vessels dilate to assure sufficient blood flow and supply of oxygen. Conversely, low carbon dioxide levels (e.g. from hyperventilation) cause the brain's blood vessels to constrict, resulting in reduced blood flow to the brain and lightheadedness. The alkalinization of blood due to hypocapnia is the mechanism by which vessels constrict (vasoconstriction); it is theorized that myofibrillar calcium sensitivity is increased in the presence of low hydrogen ion concentration.
The high pH value resulting from hyperventilation also reduces the level of available calcium (hypocalcemia), which affects the nerves and muscles, causing constriction of blood vessels and subsequent paresthesia and lightheadedness. This occurs because alkalinization of the plasma proteins (mainly albumin) increases their calcium binding affinity, thereby reducing free ionized calcium levels.
Therefore, there are two main mechanisms that contribute to the cerebral vasoconstriction that is responsible for the lightheadedness, parasthesia, and fainting often seen with hyperventilation. One mechanism is that low carbon dioxide (hypocapnia) causes increased blood pH level (respiratory alkalosis), which causes blood vessels to constrict. The other mechanism is that the decrease in hydrogen ions (alkalosis) causes decreased freely ionized blood calcium, thereby causing cell membrane instability and subsequent vasoconstriction.
Although it seems counterintuitive, breathing too much can result in a decrease in the oxygen supply to the brain. Doctors sometimes artificially induce hyperventilation after head injury to reduce the pressure in the skull, though the treatment has potential risks. [2]
Treatment
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The first step that should be taken is to treat the underlying cause of the hyperventilation. The patient should be encouraged to control his/her breathing. If this cannot be achieved, supplemental oxygen may be given to reduce tissue hypoxia. For possible behavior therapy see Treatment in Hyperventilation syndrome. Drug management is sometimes necessary. Parenteral drugs may have to be administered to reduce the patients anxiety and to slow the rate of breathing. Diazepam or midazolam are sometimes used.
References
- ^ Kenneth Baillie and Alistair Simpson. "Hyperventilation calculator". Apex (Altitude Physiology EXpeditions). Retrieved 2006-08-10. - Online interactive oxygen delivery calculator that mimics hyperventilation
- ^ Stocchetti N, Maas AI, Chieregato A, van der Plas AA (2005). "Hyperventilation in head injury: a review". Chest. 127 (5): 1812–27. doi:10.1378/chest.127.5.1812. PMID 15888864.
{{cite journal}}: CS1 maint: multiple names: authors list (link)
See also
- Hyperventilation syndrome
- Hypoventilation, too shallow or too slow breathing
- Control of respiration
- Respiratory alkalosis
- Shallow water blackout, the role of hyperventilation in some drowning incidents
- Hyperpnea
- Tachypnea