Jump to content

Endurance training: Difference between revisions

From Wikipedia, the free encyclopedia
Browse history interactively
← Previous editNext edit →
Content deleted Content added
VisualWikitext
m spelling, typos
Lordthe6 (talk | contribs)
119 edits
No edit summary
Line 10: Line 10:
Fundamental for endurance training is [[supercompensation]]. Supercompensation describes the adaptation of muscles on a previous stimulus over time.<ref>{{Cite journal|title=To the analytical theory of the supercompensation phenomenon|year=2011|doi=10.1134/S0006350911020072|last1=Brezhnev|first1=Yu. V.|last2=Zaitsev|first2=A. A.|last3=Sazonov|first3=S. V.|journal=Biophysics|volume=56|issue=2|pages=298–303|pmid=21542364|s2cid=10182848}}</ref>
Fundamental for endurance training is [[supercompensation]]. Supercompensation describes the adaptation of muscles on a previous stimulus over time.<ref>{{Cite journal|title=To the analytical theory of the supercompensation phenomenon|year=2011|doi=10.1134/S0006350911020072|last1=Brezhnev|first1=Yu. V.|last2=Zaitsev|first2=A. A.|last3=Sazonov|first3=S. V.|journal=Biophysics|volume=56|issue=2|pages=298–303|pmid=21542364|s2cid=10182848}}</ref>


Long-term endurance training induces many physiological adaptations both centrally and peripherally mediated. Central [[cardiovascular]] adaptations include decreased [[heart rate]], increased stroke volume of the heart,<ref name="stoat">"Physiology of Sport and Exercise" J.H. Wilmore, D.L. Costill, W.L. Kenney; 4th Ed, Human Kinetics 2008</ref> increased [[blood plasma]], without any major changes in [[red blood cell]] count, which reduces [[blood viscosity]] and increased [[cardiac output]] as well as total [[mitochondrial volume]] in the [[muscle fiber]]s used in the training (i.e. the thigh muscles in runners will have more mitochondria than the thigh muscles of swimmers). Changes of the hormonal regulation can be observed. Endegenous insulin secretion is increased, as well as the insulin sensitivity of muscle cells raises and glucose tolerance raises. Moderate sporting activity strengthens the immune system and reduces proneness to infection. Improved performance capacity and improvement handling of daily life needs could be observed. The aging process could be slowed down by training. Increased life expectancy of at least two years could be observed.<ref>{{Cite book|last=Neumann/Pfützner/Berbalk|url=https://books.google.com/books?id=4CVi3iZlAk4C&pg=PA280&dq=training+endurance#v=onepage&q=training%20endurance&f=false|title=Successful Endurance Training|publisher=|year=2000|isbn=1841260045|location=|pages=27–28}}</ref> Mitochondria increase in both number and size and there are similar increases in myoglobin and oxidative enzymes. Adaptations of the peripheral include [[capillarization]], that is an increase in the surface area that both the venous and arterial capillaries supply. This also allows for increased heat dissipation during strenuous exercise. The muscles heighten their [[glycogen]] and fat storing capabilities in endurance athletes in order to increase the length in time in which they can perform work. Endurance training primarily work the slow twitch (type 1) fibers and develop such fibers in their efficiency and resistance to fatigue. [[Catabolism]] also improves increasing the athletes capacity to use fat and glycogen stores as an energy source. These [[metabolic]] processes are known as [[glycogenolysis]], [[glycolysis]] and [[lipolysis]]. There is higher efficiency in oxygen transport and distribution. In recent years it has been recognized that oxidative enzymes such as succinate dehydrogenase ([[Succinate dehydrogenase|SDH]]) that enable mitochondria to break down nutrients to form [[Adenosine triphosphate|ATP]] increase by 2.5 times in well trained endurance athletes<ref name="stoat" /> In addition to SDH, [[myoglobin]] increases by 75-80% in well trained endurance athletes.<ref name="stoat" />
Long-term endurance training induces many physiological adaptations both centrally and peripherally mediated. Central [[cardiovascular]] adaptations include decreased [[heart rate]], increased stroke volume of the heart,<ref name=":0">{{Cite book|last=Kenney, W. Larry,|url=https://www.worldcat.org/oclc/889006367|title=Physiology of sport and exercise|others=Wilmore, Jack H., 1938-2014,, Costill, David L.,|isbn=978-1-4504-7767-3|edition=Sixth edition|location=Champaign, IL|oclc=889006367}}</ref> increased [[blood plasma]], without any major changes in [[red blood cell]] count, which reduces [[blood viscosity]] and increased [[cardiac output]] as well as total [[mitochondrial volume]] in the [[muscle fiber]]s used in the training (i.e. the thigh muscles in runners will have more mitochondria than the thigh muscles of swimmers). Changes of the hormonal regulation can be observed. Endegenous insulin secretion is increased, as well as the insulin sensitivity of muscle cells raises and glucose tolerance raises. Moderate sporting activity strengthens the immune system and reduces proneness to infection. Improved performance capacity and improvement handling of daily life needs could be observed. The aging process could be slowed down by training. Increased life expectancy of at least two years could be observed.<ref>{{Cite book|last=Neumann/Pfützner/Berbalk|url=https://books.google.com/books?id=4CVi3iZlAk4C&pg=PA280&dq=training+endurance#v=onepage&q=training%20endurance&f=false|title=Successful Endurance Training|publisher=|year=2000|isbn=1841260045|location=|pages=27–28}}</ref> Mitochondria increase in both number and size and there are similar increases in myoglobin and oxidative enzymes. Adaptations of the peripheral include [[capillarization]], that is an increase in the surface area that both the venous and arterial capillaries supply. This also allows for increased heat dissipation during strenuous exercise. The muscles heighten their [[glycogen]] and fat storing capabilities in endurance athletes in order to increase the length in time in which they can perform work. Endurance training primarily work the slow twitch (type 1) fibers and develop such fibers in their efficiency and resistance to fatigue. [[Catabolism]] also improves increasing the athletes capacity to use fat and glycogen stores as an energy source. These [[metabolic]] processes are known as [[glycogenolysis]], [[glycolysis]] and [[lipolysis]]. There is higher efficiency in oxygen transport and distribution. In recent years it has been recognized that oxidative enzymes such as succinate dehydrogenase ([[Succinate dehydrogenase|SDH]]) that enable mitochondria to break down nutrients to form [[Adenosine triphosphate|ATP]] increase by 2.5 times in well trained endurance athletes<ref name=":0" /> In addition to SDH, [[myoglobin]] increases by 75-80% in well trained endurance athletes.<ref name=":0" />


== Risks of excessive endurance training ==
== Risks of excessive endurance training ==

Revision as of 13:42, 29 October 2020

Endurance training is the act of exercising to increase endurance. The term endurance training generally refers to training the aerobic system as opposed to the anaerobic system. The need for endurance in sports is often predicated as the need of cardiovascular and simple muscular endurance, but the issue of endurance is far more complex. Endurance can be divided into two categories including: general endurance and specific endurance. It can be shown that endurance in sport is closely tied to the execution of skill and technique. A well conditioned athlete can be defined as, the athlete who executes his or her technique consistently and effectively with the least effort.[1] Key for measuring endurance are heart rate, power in cycling and pace in running.[2]

Endurance in sports

Endurance training is essential for a variety of endurance sports. A notable example is distance running events (800 meters upwards to marathon and ultra-marathon) with the required degree of endurance training increasing with race distance. Two other popular examples are cycling (particularly road cycling) and competitive swimming. These three endurance sports are combined in the triathlon. Other sports for which extensive amounts of endurance training are required include rowing and cross country skiing. Athletes can also undergo endurance training when their sport may not necessarily be an endurance sport in the whole sense but may still demand some endurance. For instance aerobic endurance is necessary, in varying extents, in racket sports, football, rugby, martial arts, basketball and cricket. Endurance exercise tends to be popular with non-athletes for the purpose of increasing general fitness or burning more calories to increase weight loss potential.

Physiological effects

Fundamental for endurance training is supercompensation. Supercompensation describes the adaptation of muscles on a previous stimulus over time.[3]

Long-term endurance training induces many physiological adaptations both centrally and peripherally mediated. Central cardiovascular adaptations include decreased heart rate, increased stroke volume of the heart,[4] increased blood plasma, without any major changes in red blood cell count, which reduces blood viscosity and increased cardiac output as well as total mitochondrial volume in the muscle fibers used in the training (i.e. the thigh muscles in runners will have more mitochondria than the thigh muscles of swimmers). Changes of the hormonal regulation can be observed. Endegenous insulin secretion is increased, as well as the insulin sensitivity of muscle cells raises and glucose tolerance raises. Moderate sporting activity strengthens the immune system and reduces proneness to infection. Improved performance capacity and improvement handling of daily life needs could be observed. The aging process could be slowed down by training. Increased life expectancy of at least two years could be observed.[5] Mitochondria increase in both number and size and there are similar increases in myoglobin and oxidative enzymes. Adaptations of the peripheral include capillarization, that is an increase in the surface area that both the venous and arterial capillaries supply. This also allows for increased heat dissipation during strenuous exercise. The muscles heighten their glycogen and fat storing capabilities in endurance athletes in order to increase the length in time in which they can perform work. Endurance training primarily work the slow twitch (type 1) fibers and develop such fibers in their efficiency and resistance to fatigue. Catabolism also improves increasing the athletes capacity to use fat and glycogen stores as an energy source. These metabolic processes are known as glycogenolysis, glycolysis and lipolysis. There is higher efficiency in oxygen transport and distribution. In recent years it has been recognized that oxidative enzymes such as succinate dehydrogenase (SDH) that enable mitochondria to break down nutrients to form ATP increase by 2.5 times in well trained endurance athletes[4] In addition to SDH, myoglobin increases by 75-80% in well trained endurance athletes.[4]

Risks of excessive endurance training

The potential for negative health effects from long-term, high-volume endurance training have begun to emerge in the scientific literature in recent years.[6][7][8] The known risks are primarily associated with training for and participation in extreme endurance events, and affect the cardiovascular system through adverse structural remodeling of the heart and the associated arteries, with heart-rhythm abnormalities perhaps being the most common resulting symptom.[9] Endurance exercise can also reduce testosterone levels.[10][11]

Methods and training plans

Systematic training is the key for good results. Common methods for training include periodization, intervals, hard easy, long slow distance, and in recent years high-intensity interval training.

  • The periodization method is very common and was accredited to Tudor Bompa[12] and consists of blocks of time, generally 4–12 weeks each
  • Rob Leamaker and Ray Browning promote the S.E.R.I.O.U.S method. Consisting of; Speed Training and drills, Endurance/Easy distance, Race/pace training, Intervals, Overdistance/long, slow distance, Uphill intervals/vertical training, Strength/resistance training [13]
  • MAF Training is focused on building an aerobic base, eat well, reduce stress, improve brain function [14]

Traditionally, strength training (the performance of exercises with resistance or added weight) was not deemed appropriate for endurance athletes due to potential interference in the adaptive response to the endurance elements of an athlete's training plan. There were also misconceptions regarding the addition of excess body mass through muscle hypertrophy (growth) associated with strength training, which could negatively effect endurance performance by increasing the amount of work required to be completed by the athlete. However, more recent and comprehensive research has proved that short-term (8 weeks) strength training in addition to endurance training is beneficial for endurance performance, particularly long-distance running.[15]

Literature describes following forms of endurance exercise

Form of exercise Method Goal Intensity Scope
Recovery and Compensation exercise Extensive-duration method supporting recovery lactate values below aerobic threshold no long or too extensive sessions
Extensive basic endurance exercise Extensive duration and interval technique, driving game health aspects, fortification of endurance, fat metabolism exercise lactate values clearly below aerobic threshold (50-77 %) 1–8 hours
Intensive basic endurance exercise within permanent load Intensive duration technique Improving cardio-vascular capabilities and use of glycogen load up to anaerobic threshold 77–85 % 30–120 minutes
Intensive basic endurance exercise within interval load Extensive Interval technique Improving cardio-vascular capabilities and use of glycogen Within anaerobic threshold (not further) 20–80 minutes
Competition specific intensity exercise duration technique, intensive interval technique, repetition technique, competition technique practicing specific speed within competition equal intensity within competition, within high scopes below competition intensity 50–120 % of competition scope

[16]

Devices to assess endurance fitness

The heart rate monitor is one of the relatively easy methods to assess fitness in endurance athletes. By comparing heart rate over time fitness gains can be observed when the heart rate decreases for running or cycling at a given speed. In cycling the effect of wind on the cyclists speed is difficult to subtract out and so many cyclists now use power meters built into their bicycles. The power meter allows the athlete to actually measure power output over a set duration or course and allows direct comparison of fitness progression.[17] In the 2008 Olympics Michael Phelps was aided by repeated lactate threshold measurement. This allowed his coaches to fine tune his training program so that he could recover between swim events that were sometimes several minutes apart.[18] Much similar to blood glucose for diabetes, lower priced lactate measurement devices are now available but in general the lactate measurement approach is still the domain of the professional coach and elite athlete.

See also

References

  1. ^ Michael Yessis (2008). Secrets of Russian Sports Fitness & Training. ISBN 978-0-9817180-2-6.
  2. ^ Friel, Joe (2016). The Triathlete's Training Bible: The World's Most Comprehensive Training Guide. Colorado. ISBN 9781937715441.{{cite book}}: CS1 maint: location missing publisher (link)
  3. ^ Brezhnev, Yu. V.; Zaitsev, A. A.; Sazonov, S. V. (2011). "To the analytical theory of the supercompensation phenomenon". Biophysics. 56 (2): 298–303. doi:10.1134/S0006350911020072. PMID 21542364. S2CID 10182848.
  4. ^ a b c Kenney, W. Larry,. Physiology of sport and exercise. Wilmore, Jack H., 1938-2014,, Costill, David L., (Sixth edition ed.). Champaign, IL. ISBN 978-1-4504-7767-3. OCLC 889006367. {{cite book}}: |edition= has extra text (help)CS1 maint: extra punctuation (link) CS1 maint: multiple names: authors list (link)
  5. ^ Neumann/Pfützner/Berbalk (2000). Successful Endurance Training. pp. 27–28. ISBN 1841260045.
  6. ^ Möhlenkamp S, Lehmann N, Breuckmann F, Bröcker-Preuss M, Nassenstein K, Halle M, Budde T, Mann K, Barkhausen J, Heusch G, Jöckel KH, Erbel R (200). "Running: the risk of coronary events : Prevalence and prognostic relevance of coronary atherosclerosis in marathon runners". Eur. Heart J. 29 (15): 1903–10. doi:10.1093/eurheartj/ehn163. PMID 18426850.
  7. ^ Benito B, Gay-Jordi G, Serrano-Mollar A, Guasch E, Shi Y, Tardif JC, Brugada J, Nattel S, Mont L (2011). "Cardiac arrhythmogenic remodeling in a rat model of long-term intensive exercise training" (PDF). Circulation. 123 (1): 13–22. doi:10.1161/CIRCULATIONAHA.110.938282. PMID 21173356. S2CID 7747108.
  8. ^ Wilson M, O'Hanlon R, Prasad S, Deighan A, Macmillan P, Oxborough D, Godfrey R, Smith G, Maceira A, Sharma S, George K, Whyte G (2011). "Diverse patterns of myocardial fibrosis in lifelong, veteran endurance athletes". J Appl Physiol. 110 (6): 1622–6. doi:10.1152/japplphysiol.01280.2010. PMC 3119133. PMID 21330616.
  9. ^ Potential Adverse Cardiovascular Effects From Excessive Endurance Exercise, O'Keefe et al, Mayo Clinic Proceedings, v. 87(6); June 2012
  10. ^ Bennell, KL; Brukner, PD; Malcolm, SA (September 1996). "Effect of altered reproductive function and lowered testosterone levels on bone density in male endurance athletes". British Journal of Sports Medicine. 30 (3): 205–8. doi:10.1136/bjsm.30.3.205. PMC 1332330. PMID 8889111.
  11. ^ Hackney, AC (October 2008). "Effects of endurance exercise on the reproductive system of men: the 'exercise-hypogonadal male condition'". Journal of Endocrinological Investigation. 31 (10): 932–8. doi:10.1007/bf03346444. PMID 19092301. S2CID 4706924.
  12. ^ The Triathlete's Training Bible, Joe Friel, 2nd ed, p. 28, Velo Press, 2004
  13. ^ Browning; Sleamaker, Rob; Ray (1996). Serious training for endurance Athletes. ISBN 0873226445.{{cite book}}: CS1 maint: multiple names: authors list (link)
  14. ^ Maffetone, Philip (2010). The big Book of endurance Training and Racing. ISBN 9781616080655.
  15. ^ Beattie, K; Kenny, IC; Lyons, M; Carson, BP (2014). "The Effect of Strength Training on Performance in Endurance" (PDF). Sports Medicine. 44 (6): 845–65. doi:10.1007/s40279-014-0157-y. hdl:10344/4182. PMID 24532151. S2CID 16340630.
  16. ^ Optimiertes Ausdauertraining. Aachen: Georg Neumann, Arndt Pfützner, Anneliese Berbalk. 1998. ISBN 3898996158.
  17. ^ The Triathlete's Training Bible, Joe Friel, 2nd ed, pp 64-65, Velo Press, 2004
  18. ^ "2008 Olympic Coverage"
Retrieved from "https://en.wikipedia.org/w/index.php?title=Endurance_training&oldid=986046231"