Tachycardia
| Tachycardia | |
|---|---|
| Specialty | Cardiology  |
Tachycardia is a heart rate that exceeds the normal range. In general, a resting heart rate over 100 beats per minute is accepted as tachycardia. Tachycardia can be caused by various factors that often are benign. However, tachycardia can be dangerous, depending on the speed and type of rhythm. Note that, if it is pathological, a tachycardia is more correctly defined as a tachyarrhythmia.
Etymology
Tachycardia comes from the Greek ταχύς tachys, "quick, rapid" and καρδία, kardia, "heart".
Definition
The upper threshold of a normal human resting heart rate is based upon age. Tachycardia for different age groups is as listed below:[1]
- 1–2 days: Tachycardia > 159 beats per minute (bpm)
- 3–6 days: Tachycardia >166 bpm.
- 1–3 weeks: Tachycardia >182 bpm
- 1–2 months: Tachycardia >179 bpm
- 3–5 months: Tachycardia >186 bpm
- 6–11 months: Tachycardia >169 bpm
- 1–2 years: Tachycardia >151 bpm
- 3–4 years: Tachycardia >137 bpm
- 5–7 years: Tachycardia >133 bpm
- 8–11 years: Tachycardia >130 bpm
- 12–15 years: Tachycardia >119 bpm
- >15 years – adult: Tachycardia >100* bpm
Heart rate is often correlated with clinical picture, in sepsis >90 bpm is considered tachycardia.
When the heart beats excessively or rapidly, the heart pumps less efficiently and provides less blood flow to the rest of the body, including the heart itself. The increased heart rate also leads to increased work and oxygen demand by the heart, which can lead to rate related ischemia.[2]
Differential diagnosis
An electrocardiogram (ECG) is used to classify the type of tachycardia. They may be classified into narrow and wide complex based on the QRS complex.[3] Presented in the order of most to least common they are:[3]
Narrow complex
- Sinus tachycardia, which originates from the sino-atrial (SA) node, near the base of the superior vena cava.
- Atrial fibrillation
- Atrial flutter
- AV nodal reentrant tachycardia
- Accessory pathway mediated tachycardia
- Atrial tachycardia
- Multifocal atrial tachycardia
- Junctional tachycardia
Wide complex
- Ventricular tachycardia, any tachycardia that originates in the ventricles.
- Any narrow complex tachycardia combined with a problem with the conduction system of the heart, often termed "supraventricular tachycardia with aberrancy."
- A narrow complex tachycardia with an accessory conduction pathway, often termed "supraventricular tachycardia with pre-excitation" (e.g. Wolff–Parkinson–White syndrome).
- Pacemaker-tracked or pacemaker-mediated tachycardia.
Tachycardias may be classified as either narrow complex tachycardias (supraventricular tachycardias) or wide complex tachycardias. Narrow and wide refer to the width of the QRS complex on the ECG. Narrow complex tachycardias tend to originate in the atria, while wide complex tachycardias tend to originate in the ventricles. Tachycardias can be further classified as either regular or irregular.
Sinus
The body has several feedback mechanisms to maintain adequate blood flow and blood pressure. If blood pressure decreases, the heart beats faster in an attempt to raise it. This is called reflex tachycardia. This can happen in response to a decrease in blood volume (through dehydration or bleeding), or an unexpected change in blood flow. The most common cause of the latter is orthostatic hypotension (also called postural hypotension). Fever, hyperventilation, diarrhea and severe infections can also cause tachycardia, primarily due to increase in metabolic demands.
An increase in sympathetic nervous system stimulation causes the heart rate to increase, both by the direct action of sympathetic nerve fibers on the heart and by causing the endocrine system to release hormones such as epinephrine (adrenaline), which have a similar effect. Increased sympathetic stimulation is usually due to physical or psychological stress. This is the basis for the so-called fight-or-flight response, but such stimulation can also be induced by stimulants such as ephedrine, amphetamines or cocaine. Certain endocrine disorders such as pheochromocytoma can also cause epinephrine release and can result in tachycardia independent nervous system stimulation. Hyperthyroidism can also cause tachycardia.[4] The upper limit of normal rate for sinus tachycardia is thought to be 220 bpm minus age.
Ventricular
Ventricular tachycardia (VT or V-tach) is a potentially life-threatening cardiac arrhythmia that originates in the ventricles. It is usually a regular, wide complex tachycardia with a rate between 120 and 250 beats per minute.
Both of these rhythms normally last for only a few seconds to minutes (paroxysmal tachycardia), but if VT persists it is extremely dangerous, often leading to ventricular fibrillation.
Supraventricular
This is a type tachycardia that originates from above the ventricles, such as the atria. It is sometimes known as paroxysmal atrial tachycardia (PAT). Several types of supraventricular tachycardia are known to exist.
Atrial fibrillation
Atrial fibrillation is one of the most common cardiac arrhythmias. In general, it is an irregular, narrow complex rhythm. However, it may show wide QRS complexes on the ECG if a bundle branch block is present. At high rates, the QRS complex may also become wide due to the Ashman phenomenon. It may be difficult to determine the rhythm's regularity when the rate exceeds 150 beats per minute. Depending on the patient's health and other variables such as medications taken for rate control, atrial fibrillation may cause heart rates that span from 50 to 250 beats per minute (or even higher if an accessory pathway is present). However, new onset atrial fibrillation tends to present with rates between 100 and 150 beats per minute.
AV nodal reentrant tachycardia
AV nodal reentrant tachycardia (AVNRT) is the most common reentrant tachycardia. It is a regular narrow complex tachycardia that usually responds well to the Valsalva maneuver or the drug adenosine. However, unstable patients sometimes require synchronized cardioversion. Definitive care may include catheter ablation.
AV reentrant tachycardia
AV reentrant tachycardia (AVRT) requires an accessory pathway for its maintenance. AVRT may involve orthodromic conduction (where the impulse travels down the AV node to the ventricles and back up to the atria through the accessory pathway) or antidromic conduction (which the impulse travels down the accessory pathway and back up to the atria through the AV node). Orthodromic conduction usually results in a narrow complex tachycardia, and antidromic conduction usually results in a wide complex tachycardia that often mimics ventricular tachycardia. Most antiarrhythmics are contraindicated in the emergency treatment of AVRT, because they may paradoxically increase conduction across the accessory pathway.
Junctional tachycardia
Junctional tachycardia is an automatic tachycardia originating in the AV junction. It tends to be a regular, narrow complex tachycardia and may be a sign of digitalis toxicity.
Management
The management of tachycardia depends on its type (wide complex versus narrow complex), whether or not the person is stable or unstable, and whether the instability is due to the tachycardia.[3] Unstable means that either important organ functions are affected or cardiac arrest is about to occur.[3]
Stable
In those that are stable, treatment is determined by the exact electrocardiogram findings: wide versus narrow complex, regular versus irregular heart rate, and whether the QRS is monomorphic or polymorphic.
Unstable
In those that are unstable with a narrow complex tachycardia, intravenous adenosine may be attempted.[3] In all others immediate cardioversion is recommended.[3]
Nonmedical & herbal alternatives
In the instance medical care is not available several alternatives for managing tachycaridia are presented. The main concern is that the heart should not stop fully or quiver (Fibrillation). Hawthorne and hibiscus are considered cardiotonics in some schools of traditional medicine. D-limonene is an agonist at adenosine receptors, and may provide similar relief through the oral route as intravenous adenosine.
In the case that no drug or herbal alternatives are available, an increase in physical activity can help to 'load' the heart by increasing blood flow to the extremities; and coordinated action of the diaphram and extremities also assists in pumping blood. If the instance of tachycardia is not pathological, increasing physical activity may help stave off fibrillation and decrease acute stress on cardiac muscle.
References
- ^ Custer JW, Rau RE, eds. Johns Hopkins: The Harriet Lane Handbook. 18th ed. Philadelphia, PA: Mosby Elsevier Inc; 2008. Data also available through eMedicine: Pediatrics, Tachycardia.
- ^ Harrison's Principles of Internal Medicine, 17th Edition
- ^ a b c d e f Neumar RW, Otto CW, Link MS; et al. (November 2010). "Part 8: adult advanced cardiovascular life support: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care". Circulation. 122 (18 Suppl 3): S729–67. doi:10.1161/CIRCULATIONAHA.110.970988. PMID 20956224.
{{cite journal}}: Explicit use of et al. in:|author=(help)CS1 maint: multiple names: authors list (link) - ^ Barker RL, Burton JR, Zieve, PD eds. Principles of Ambulatory Medicine. Sixth Edition. Philadelphia, PA: Lippinocott, Wilkins & Williams 2003