Myocardiodystrophy

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Myocardiodystrophy
Classification and external resources

Myocardiodystrophy is a type of noninflammatory heart disease that affects the myocardium. It is characterized by cardiac pain that occurs in the morning and that cannot be stopped even with normal treatments of nitroglycerin, along with other occurrences of heart rhythm disturbances, dyspnea, and various types of heart failure.[1][2]

Cause[edit]

There are multiple originations for myocardiodystrophy. Alcohol intoxication can originate the creation of it, along with making it worse if consumption continues over a long period of time. Different types of obesity can also originate the disease,[3] along with other endocrine diseases, infections, and various types of neurological pathologies.[1]

References[edit]

  1. ^ a b Myocardiodystrophy on CALMED[unreliable medical source?]
  2. ^ Dorofeyeva EE, Dorofeyev AE (November 2004). "Biochemical and heart adaptations to physical training and supplementation with amino acids". Journal of Strength and Conditioning Research. 18 (4): 738–40. PMID 15574076. doi:10.1519/12922.1. 
  3. ^ Sinitsyn PD, Kholodova EA (October 1983). "[Diagnosis and pathogenesis of diabetic myocardiodystrophy]". Kardiologiia (in Russian). 23 (10): 35–8. PMID 6645168. 

Further reading[edit]

  • Gnatiuk MS; Slivka IuI; Gnatiuk LA (1998). "[Secretory activity of atrial cardiomyocytes in adrenaline myocardiodystrophy]". Patologicheskaia Fiziologiia I Èksperimental'naia Terapiia (in Russian) (2): 36–7. PMID 9633198. 
  • Temirova KV, Kurlygina LA, Zavodskaia IS, Novikova NA (September 1976). "[Energy metabolism and myocardial function in myocardiodystrophy]". Kardiologiia (in Russian). 16 (9): 98–102. PMID 1011536. 
  • Tret'iakov SV, Loseva MI, Shpagina LA (1997). "[The characteristics of cardiac diastolic function in myocardiodystrophy in persons subjected to aromatic hydrocarbon exposure]". Terapevticheskiĭ Arkhiv (in Russian). 69 (1): 47–9. PMID 9163050. 
  • Iakimenko-Vasiutina EA (August 1990). "[The treatment of women with dyshormonal myocardiodystrophy]". Vrachebnoe Delo (in Russian) (8): 65–7. PMID 2256296. 
  • Eliseev, V. E.; Slobodskaya, V. V.; Il'in, G. I.; Kostin, É. D. (1985). "Influence of Riboxine, uridine, uridine-5'-monophosphate, and guanosine on experimental myocardial dystrophy". Pharmaceutical Chemistry Journal. 19 (6): 406–8. doi:10.1007/BF00766947. Archived from the original on 2011-07-14. 
  • Goncharova EV, Govorin AV (2008). "[Changes in fatty acids content in blood red cells of patients with iron-deficiency anemia treated with sorbifer and mildronate]". Terapevticheskiĭ Arkhiv (in Russian). 80 (6): 65–8. PMID 18655480.