|This is the talk page for discussing improvements to the Pulmonary artery article.|
|WikiProject Anatomy||(Rated Start-class, Mid-importance)|
|The content of Pulmonary artery pressure was merged into Pulmonary artery on 28 September 2013. That page now redirects here. For the contribution history and old versions of the redirected page, please see ; for the discussion at that location, see its talk page.|
Does anyone else see weird rendering problems on this page? A couple images overlap unless you have really wide browser windows.
Yes, I just noticed this too. Bug needs Fixing
- I would propose the contents of Pulmonary wedge pressure to be suitably copied into this article but I feel the other topic bears the significance of its own name and so that article should be maintained and expanded but not deleted. DiptanshuTalk 17:25, 26 December 2013 (UTC)
Pulmonary artery occlusion pressure or wedge pressure not the same as pulmonary capillary wedge pressure
The terms PAWP and pulmonary artery occlusion pressure (PAOP) are used interchangeably and refer to the same measurement obtained from the tip of a PAC following balloon inflation and flotation to the wedged position. As already discussed, PAWP and PAOP are used as indirect estimates of mean LAP. In contrast, the hydrostatic pressure in the pulmonary capillaries is a different pressure that must exceed Left Atrial Pressure (LAP) to maintain antegrade blood flow through the lungs. This pulmonary capillary pressure must not be confused with PAWP or LAP, nor should these terms be used interchangeably. Continued use of the phrase “pulmonary capillary wedge pressure” to mean PAWP or PAOP has perpetuated misconceptions about these measurements. Although the magnitude of the difference between pulmonary capillary pressure and PAWP is generally small, it can increase markedly when resistance to flow in the pulmonary veins is elevated. In most situations, the major component of pulmonary vascular resistance occurs at the precapillary, pulmonary arteriolar level. However, rare conditions like pulmonary veno-occlusive disease may cause a marked increase in postcapillary resistance to flow within the pulmonary veins. A similar situation arises in other conditions that disproportionately increase pulmonary venous resistance, such as central nervous system injury, acute lung injury, hypovolemic shock, endotoxemia, and norepinephrine infusion. Under these conditions, measurement of PAWP will underestimate pulmonary capillary pressure substantially and thereby underestimate the risk of hydrostatic pulmonary edema. Although pulmonary capillary pressure may be measured at the bedside by analyzing the decay in pulmonary artery pressure (PAP) following pulmonary artery catheter (PAC) balloon inflation, these techniques have not been adopted widely in clinical practice. To avoid confusion, the phrase “pulmonary capillary wedge pressure” should be abandoned because it is imprecise and misleading.
http://web.squ.edu.om/med-Lib/MED_CD/E_CDs/anesthesia/site/content/v03/030280r00.HTM — Preceding unsigned comment added by Cheryldrn (talk • contribs) 16:37, 9 March 2014 (UTC)