Erection: Difference between revisions
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{{about|penile erection|clitoral erection|Clitoral erection|the erection of a building|Construction}} |
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[[Image:Erection_Homme.jpg|thumb|right|A penis flaccid (left) and erect (right)]] |
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==Physiology== |
==Physiology== |
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[[File:Erection Development.jpg|thumb|200px|right|Composite image showing the development of a penile erection]] |
[[File:Erection Development.jpg|thumb|200px|right|Composite image showing the development of a penile erection]] |
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Penile erection occurs when two tubular structures that run the length of the penis, the [[Corpus cavernosum penis|corpora cavernosa]], become engorged with venous blood. This may result from any of various [[physiology|physiological]] stimuli, also known as sexual stimulation and [[sexual arousal]]. The [[corpus spongiosum]] is a single tubular structure located just below the corpora cavernosa, which contains the [[urethra]], through which [[urine]] and [[semen]] pass during [[urination]] and [[ejaculation]], respectively. This may also become slightly [[engorged]] with blood, but less so than the corpora cavernosa. |
Penile erection occurs when two tubular structures that run the length of the penis, the [[Corpus cavernosum penis|corpora cavernosa]], become engorged with venous blood. This may result from any of various [[physiology|physiological]] stimuli, also known as sexual stimulation and [[sexual arousal]]. The [[corpus spongiosum]] is a single |
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#REDIRECT [[Penile Erection]] |
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tubular structure located just below the corpora cavernosa, which contains the [[urethra]], through which [[urine]] and [[semen]] pass during [[urination]] and [[ejaculation]], respectively. This may also become slightly [[engorged]] with blood, but less so than the corpora cavernosa. |
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===During sexual activity=== |
===During sexual activity=== |
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Revision as of 08:27, 16 April 2011
Penile erection is a physiological phenomenon where the penis becomes enlarged and firm. Penile erection is the result of a complex interaction of psychological, neural, vascular and endocrine factors, and is usually, though not exclusively, associated with sexual arousal. Penile erection can also occur due to a full urinary bladder. In some males, erection can occur spontaneously at any time of day, and is known as nocturnal penile tumescence when occurring during rapid eye movement sleep.
Physiology
Penile erection occurs when two tubular structures that run the length of the penis, the corpora cavernosa, become engorged with venous blood. This may result from any of various physiological stimuli, also known as sexual stimulation and sexual arousal. The corpus spongiosum is a single
- REDIRECT Penile Erection
tubular structure located just below the corpora cavernosa, which contains the urethra, through which urine and semen pass during urination and ejaculation, respectively. This may also become slightly engorged with blood, but less so than the corpora cavernosa.
During sexual activity
The swelling, hardening and enlargement of the penis enables sexual intercourse. The scrotum may also become tightened during an erection. In many cases, the foreskin automatically and gradually retracts, exposing the glans, but some males may have to manually retract their foreskin.
After a male has ejaculated during a sexual encounter or masturbation, the erection usually ends, but this may take time depending on the length and thickness of the penis.[1]
Autonomic control
In the presence of mechanical stimulation, erection is initiated by the parasympathetic division of the autonomic nervous system (ANS) with minimal input from the central nervous system. Parasympathetic branches extend from the sacral plexus into the arteries supplying the erectile tissue; upon stimulation, these nerve branches release acetylcholine, which, in turn causes release of nitric oxide from endothelial cells in the trabecular arteries.[2] Nitric oxide diffuses to the smooth muscle of the arteries (called trabecular smooth muscle[3]), acting as a vasodilating agent. The arteries dilate, filling the corpora spongiosum and cavernosa with blood. The ischiocavernosus and bulbospongiosus muscles also compress the veins of the corpora cavernosa, limiting the venous drainage of blood.[4] Erection subsides when parasympathetic stimulation is discontinued; baseline stimulation from the sympathetic division of the ANS causes constriction of the penile arteries, forcing blood out of the erectile tissue.[5] The cerebral cortex can initiate erection in the absence of direct mechanical stimulation (in response to visual, auditory, olfactory, imagined, or tactile stimuli) acting through erectile centers in the lumbar and sacral regions of the spinal cord. The cortex can suppress erection even in the presence of mechanical stimulation, as can other psychological, emotional, and environmental factors. The opposite term is detumescence.
Shape and size
Although many erect penises point upwards, it is common and normal for the erect penis to point nearly vertically upwards or nearly vertically downwards or even horizontally straight forward, all depending on the tension of the suspensory ligament that holds it in position. An erect penis can also take on a number of different shapes, ranging from a straight tube to a tube with a curvature up or down or to the left or right. An increase in penile curvature can be caused by Peyronie's disease. This may cause physical and psychological effects for the affected individual, which could include erectile dysfunction or pain during erection. Treatments include oral medication (such as Colchicine) or surgery, which is most often reserved as a last resort.
The following table shows how common various erection angles are for a standing male. In the table, zero degrees is pointing straight up against the abdomen, 90 degrees is horizontal and pointing straight forward, while 180 degrees would be pointing straight down to the feet. An upward pointing angle is most common.
| Angle (º) | Percent |
|---|---|
| 0–30 | 5 |
| 30–60 | 30 |
| 60–85 | 31 |
| 85–95 | 10 |
| 95–120 | 20 |
| 120–180 | 5 |
Generally, the size of an erect penis is fixed throughout post-pubescent life. Its size may be increased by surgery,[7] although penile enlargement is controversial, and a majority of men were "not satisfied" with the results, according to one study.[8]
Erectile dysfunction
Erectile dysfunction (also known as ED or "(male) impotence") is a sexual dysfunction characterized by the inability to develop and/or maintain an erection.[9][10] The study of erectile dysfunction within medicine is known as andrology, a sub-field within urology.[11]
Erectile dysfunction can occur due to both physiological and psychological reasons, most of which are amenable to treatment. Common physiological reasons include diabetes, kidney disease, chronic alcoholism, multiple sclerosis, atherosclerosis, vascular disease, and neurologic disease which collectively account for about 70 percent of ED cases. Some drugs used to treat other conditions, such as lithium and paroxetine, may cause erectile dysfunction.[10][12]
Erectile dysfunction, tied closely as it is to cultural notions of potency, success and masculinity, can have devastating psychological consequences including feelings of shame, loss or inadequacy;[13] There is a strong culture of silence and inability to discuss the matter. In fact, around one in ten men will experience recurring impotence problems at some point in their lives.[14]
See also
.svg){kind=small}
- Clitoral erection
- Death erection
- Erectores pilorum
- Nipple erection
- Nocturnal penile tumescence
- Priapism
- Sexual function
References
- ^ Harris, Robie H. (et al.), It's Perfectly Normal: Changing Bodies, Growing Up, Sex And Sexual Health. Boston, 1994. (ISBN 1-56402-199-8)
- ^ wiley.com > Viagra function image Retrieved on Mars 11, 2010
- ^ APDVS > 31. Anatomy and Physiology of Normal Erection Retrieved on Mars 11, 2010
- ^ Moore, Keith (2007). Essential Clinical Anatomy, Third Edition. Lippincott Williams & Wilkins. p. 265. ISBN 0-7817-6274-X.
{{cite book}}: Unknown parameter|coauthors=ignored (|author=suggested) (help) - ^ Drake, Richard, Wayne Vogl and Adam Mitchell, Grey's Anatomy for Students. Philadelphia, 2004. (ISBN 0-443-06612-4
- ^ Sparling J (1997). "Penile erections: shape, angle, and length". Journal of Sex & Marital Therapy. 23 (3): 195–207. PMID 9292834.
- ^ Li CY, Kayes O, Kell PD, Christopher N, Minhas S, Ralph DJ (2006). "Penile suspensory ligament division for penile augmentation: indications and results". Eur. Urol. 49 (4): 729–733. doi:10.1016/j.eururo.2006.01.020. PMID 16473458.
{{cite journal}}: CS1 maint: multiple names: authors list (link) - ^ "Most Men Unsatisfied With Penis Enlargement Results". Fox News. 2006-02-16. Retrieved 2008-08-17.
- ^ Milsten, Richard (et al.), The Sexual Male. Problems And Solutions. London, 2000. (ISBN 0-393-32127-4)
- ^ a b Sadeghipour H, Ghasemi M, Ebrahimi F, Dehpour AR (2007). "Effect of lithium on endothelium-dependent and neurogenic relaxation of rat corpus cavernosum: role of nitric oxide pathway". Nitric Oxide. 16 (1): 54–63. doi:10.1016/j.niox.2006.05.004. PMID 16828320.
{{cite journal}}: CS1 maint: multiple names: authors list (link) - ^ Williams, Warwick, It's Up To You: Overcoming Erection Problems. London, 1989. (ISBN 0-7225-1915-X)
- ^ Sadeghipour H, Ghasemi M, Nobakht M, Ebrahimi F, Dehpour AR (2007). "Effect of chronic lithium administration on endothelium-dependent relaxation of rat corpus cavernosum: the role of nitric oxide and cyclooxygenase pathways". BJU Int. 99 (1): 177–182. doi:10.1111/j.1464-410X.2006.06530.x. PMID 17034495.
{{cite journal}}: CS1 maint: multiple names: authors list (link) - ^ Tanagho, Emil A. (et al.), Smith's General Urology. London, 2000. (ISBN 0-8385-8607-4)
- ^ NHS Direct – Health encyclopaedia -Erectile dysfunction
