Cannabis withdrawal: Difference between revisions
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Significantly, evidence indicates that withdrawal symptoms are alleviated when cannabis users resume using cannabis after a period of abstinence<ref name="Budney, A.J. 2001"/> and recent laboratory research has focused on the role of brain chemistry in cannabis dependence. |
Significantly, evidence indicates that withdrawal symptoms are alleviated when cannabis users resume using cannabis after a period of abstinence<ref name="Budney, A.J. 2001"/> and recent laboratory research has focused on the role of brain chemistry in cannabis dependence. |
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In comparitive to other 'drugs of dependance' cannabis has a relative non-existant psychical withdrawl period, rather majoritively psychological. The important distinction in these terms is that whilst drugs mroe commonly associate to intense withdrawls (Methamphetamine, Heroin, Opiates etc) the active component of Cannabis, THC (tetrahydrocannabinol) is a primarily psychoactive compound. The withdrawl effects from cannabis are often largely dependant on the nature of the users level of addiction and the mental state upon entering into rehabilitation. A commonly associate side affect from long-term use of cannabis is the possibility of inducing minor to severe cases of psychosis with the potential to lead to schizophrenia or further psychotic episodes. |
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In process of rehabilitation often the primary means of overcoming a cannabis addiction is more prevelant to overcoming a habbitual addiction to the route of administration. Commonly chronic users will find it more difficult to overcome an addiction to their route of administration (ie: finding it difficult to cease use of a bong or pipe) moreso than the actual substance itself. |
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In its natural form Delta 9 THC acts as an anti-anxietal, and often the nature of anti-anxietal contributes to the effects of withdrawl inducing the user into further anxious or paranoid mental states. The psychological effects of cannabis use in withdrawl are similar yet significantly less than that of similar 'happy' drugs such as Methamphetamines. Whilst Cannabis itself does not replace ceretonin in the brain, it does have a direct effect in the standard or 'natural' levels presents in the brain. predominantly the effects of long term cannabis use can be reversed gathering the user has a strong enough mental state to withdraw without residual effect. |
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==See also== |
==See also== |
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* [[Long-term effects of cannabis]] |
* [[Long-term effects of cannabis]] |
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Revision as of 07:29, 3 March 2012
| Cannabis withdrawal |
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Cannabis withdrawal is a form of withdrawal associated with the substance cannabis. It is included in the proposed revision of DSM-5.[1]
At one time cannabis was considered a drug that had no withdrawal symptoms because users did not display symptoms similar to those withdrawing from alcohol or opiates. Contrary to this, experimental research supports reports of users who relate evidence of heavy cannabis use producing comparatively mild psychological withdrawal symptoms.[2][3]
Kouri and Pope examined withdrawal symptoms over 28 days abstinence from cannabis,[4] while Budney et al. looked at a time period of abstinence of 45 days.[3] Their study assessed withdrawal symptoms among chronic cannabis users who were assessed daily on various symptoms while on a hospital ward for 28 days. They rated mood, anxiety, depression and irritability and compared them to those of two control groups of abstinent former heavy cannabis users and non-users of cannabis. Chronic cannabis users showed decreases in mood and appetite and increases in irritability and anxiety and their scores on the Hamilton Rating Scale for Depression scale increased. Both studies used urinalysis to ensure abstinence, and showed that withdrawal symptoms began within 1–3 days of abstinence and lasted for 10–14 days.[2][3][4] According to Budney et al., the withdrawal syndrome associated with cannabis use is similar to that for tobacco but of lesser magnitude than withdrawal from other drugs like opiates or alcohol.[3]
Significantly, evidence indicates that withdrawal symptoms are alleviated when cannabis users resume using cannabis after a period of abstinence[2] and recent laboratory research has focused on the role of brain chemistry in cannabis dependence.
In comparitive to other 'drugs of dependance' cannabis has a relative non-existant psychical withdrawl period, rather majoritively psychological. The important distinction in these terms is that whilst drugs mroe commonly associate to intense withdrawls (Methamphetamine, Heroin, Opiates etc) the active component of Cannabis, THC (tetrahydrocannabinol) is a primarily psychoactive compound. The withdrawl effects from cannabis are often largely dependant on the nature of the users level of addiction and the mental state upon entering into rehabilitation. A commonly associate side affect from long-term use of cannabis is the possibility of inducing minor to severe cases of psychosis with the potential to lead to schizophrenia or further psychotic episodes.
In process of rehabilitation often the primary means of overcoming a cannabis addiction is more prevelant to overcoming a habbitual addiction to the route of administration. Commonly chronic users will find it more difficult to overcome an addiction to their route of administration (ie: finding it difficult to cease use of a bong or pipe) moreso than the actual substance itself.
In its natural form Delta 9 THC acts as an anti-anxietal, and often the nature of anti-anxietal contributes to the effects of withdrawl inducing the user into further anxious or paranoid mental states. The psychological effects of cannabis use in withdrawl are similar yet significantly less than that of similar 'happy' drugs such as Methamphetamines. Whilst Cannabis itself does not replace ceretonin in the brain, it does have a direct effect in the standard or 'natural' levels presents in the brain. predominantly the effects of long term cannabis use can be reversed gathering the user has a strong enough mental state to withdraw without residual effect.
See also
References
- ^ "Proposed Revision | APA DSM-5". Retrieved 2010-04-26.
- ^ a b c Attention: This template ({{cite pmid}}) is deprecated. To cite the publication identified by PMID 11576029, please use {{cite journal}} with
|pmid=11576029instead. - ^ a b c d Attention: This template ({{cite pmid}}) is deprecated. To cite the publication identified by PMID 12943018, please use {{cite journal}} with
|pmid=12943018instead. - ^ a b Attention: This template ({{cite pmid}}) is deprecated. To cite the publication identified by PMID 11127420, please use {{cite journal}} with
|pmid=11127420instead.
External links
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