This user helped "Amphetamine" become a good article on November 30, 2013.
This user helped "Methamphetamine" become a good article on January 27, 2014.
This user drew "File:TAAR1 Dopamine.svg", a valued image on Wikimedia Commons within the scope "Category:Amphetamine, pharmacodynamics." (March 19, 2014)‎
This user drew "File:TAAR1 Dopamine.svg", a quality image on Wikimedia Commons. (March 30, 2014)
This user helped "Adderall" become a good article on December 3, 2014.
This user helped "Amphetamine" become a featured article on January 14, 2015.
This user has AutoWikiBrowser permissions on the English Wikipedia.


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Reliability of Wikipedia
Semi-related to writing Wikipedia: [1][2]

I have an academic background in mathematics, statistics, finance, and economics. I have a general interest in and deep knowledge of molecular neuropharmacology as well, primarily in relation to neuropsych disorders and basically anything remotely related to monoamine neurotransmission (e.g., signal transduction in associated pathways, neural pathway interactions, and pharmacodynamics/pharmacogenomics of relevant drugs).
I generally only edit articles on neurology, psychology, pharmacology, cell signaling, or medical topics. I also spend a fair amount of time gnoming WP and continuously donate my 2¢ to WT:MED since I'm generous like that.

Animal Cell Unannotated.svg This user is a member of the Molecular & Cell Bio. WikiProject.
Rod of asclepius.png This user is a member of WikiProject Medicine.
Penicillin core.svg This user is a member of WikiProject Pharmacology.
Brain logo.svg This user contributes to the Neuroscience WikiProject.
Plato-raphael.jpg This user has published peer-reviewed articles in academic journals.
FSM.svg This user is a devout worshipper of His Noodliness.
Wikipedia-logo-v2.svg This user is severely addicted to Wikipedia.
Book3.svg This user is an autodidact in subjects that he never studied at a university.
*.* This user has ADOS:
Attention Deficit OOH Shiny
Red barnstar.png This user is a recipient of the
Editor of the Week award.
Tentative unprioritized future editing plans
WP:MCB articles
Intent WP:NEURO and miscellaneous
Nitazoxanide Featured article Neurobiological effects of physical exercise Featured article
Methamphetamine Featured article Reward system
ADHD Featured article Rostromedial tegmental nucleus
3-Hydroxyisovaleric acid Featured article Eisbrecher
Trace amine
My contributions to Wikipedia
  1. Amphetamine - (Page statistics) (Also: Adderall and dextroamphetamine mostly through transclusions)
  2. Methamphetamine - (Page statistics)
  3. Beta-Hydroxy beta-methylbutyric acid‎‎ - (Page statistics)
  4. Neurobiological effects of physical exercise - in progress (Page statistics)
  • Examples of images I've drawn and wiki-annotated
  1. {{Psychostimulant addiction}}File:ΔFosB.svg
  2. {{Amphetamine pharmacodynamics}}Valued image within the scope "Category:Amphetamine, pharmacodynamics"Quality imageFile:TAAR1 Dopamine.svg
  3. {{Amphetamine pharmacokinetics}} – File:Amph Pathway.png
  4. {{Catecholamine and trace amine biosynthesis}} – File:Catecholamine and trace amine biosynthesis.png
  • The image annotation template that I coded for overlaying wikitext:
{{Annotated image 4}}

Lastly, the most ridiculous article that I've come come across so far:

The result of
The image above contains clickable links
...that I created.

Cross-wiki userpage links[edit]

Links to my userpages on other Wikimedia projects where I contribute content:

Maintained articles and templates[edit]

Pages that I actively monitor or frequently edit


For myself[edit]

Medicine-related article alerts[edit]

Medicine article alerts

Articles for deletion
Proposed deletions
  • 29 Aug 2016 – Abdullah A. Al Abdulgader (talk · edit · hist) was PRODed by Jerodlycett (t · c): He does not in himself seems notable. There is also no citation for most of it and the citation given for his education does not support it.
  • 27 Aug 2016 – Neurochemical flux dysregulation (talk · edit · hist) was PRODed by PamD (t · c): No evidence of notability- no ghits for the term, only ref is to raw data table.
  • 25 Aug 2016 – Mary Joy (talk · edit · hist) was PRODed by Project Osprey (t · c): Not notable per WP:CORP. News articles focuses on the materials they distribute (which is distinct from produce) but there's no real coverage of the company itself. They appear to have stopped trading. Outfits such as this tend to appear and ...
Categories for discussion
Redirects for discussion
Featured article candidates
Good article nominees
Featured article reviews
Requests for comments
Peer reviews

Citations I plan to use[edit]

Amph male sexual function[1] - long-term cerebellar ADHD stimulant effects


To read through later:

Links, pre-formatted wikitext, and other content[edit]

Preformatted templates[edit]

{{talk header}}
{{WPBS      |  1=
{{WPPHARM   |  class =  | importance =  }}
{{WPMED     |  class =  | importance =  }}
{{WPNEURO   |  class =  | importance =  }}
{{WPMCB     |  class =  | importance =  }}
{{Chemicals |  class =  | importance =  }}
{{Reliable sources for medical articles}}
Preformatted template for MN text

<ref name="NHM-#">{{cite book | vauthors = Malenka RC, Nestler EJ, Hyman SE | editor = Sydor A, Brown RY | title = Molecular Neuropharmacology: A Foundation for Clinical Neuroscience | year = 2009 | publisher = McGraw-Hill Medical | location = New York | isbn = 9780071481274 | pages = – | edition = 2nd | chapter = Chapter <!--#:XYZ--> | quote= }}</ref>

Useful links[edit]

Useful page/syntax references[edit]

  • Redirect templates:
    • For redirects that result from a merge, use: {{tpr|target article name|merge=yes}} (Template:Talk page of redirect shortcut)
    • Category parameters for {{Redr}} are listed in the navbox below:
Project-space links
Wikipedia:WikiProject Medicine
Recent changes in WP:Medicine
Articles and their talkpages:

Not mainspace:

 Top   High   Mid   Low   NA   ???  Total
90 1,005 8,871 17,293 7,163 1,820 36,242
List overview · Lists updated: 2015-07-15 · This box: view · talk
Amusing pages

Miscellaneous links and article content[edit]

Follow-up request link for FAC coordinators: Special:diff/735661620/735781345

Editor tools
  1. Pageviews tool
  2. Wikichecker
  3. Edit history statistics
  4. Find images (semi-crappy tool)
  5. Article contributor ranking
Other stuff


  1. ^ De Luca LA, Menani JV, Johnson AK (2014). Neurobiology of Body Fluid Homeostasis: Transduction and Integration. Boca Raton: CRC Press/Taylor & Francis. ISBN 9781466506930. Retrieved 23 July 2016. 
  2. ^ Kennedy DO (January 2016). "B Vitamins and the Brain: Mechanisms, Dose and Efficacy-A Review". Nutrients. 8 (2). doi:10.3390/nu8020068. PMC 4772032free to read. PMID 26828517. Furthermore, evidence from human research clearly shows both that a significant proportion of the populations of developed countries suffer from deficiencies or insufficiencies in one or more of this group of vitamins, and that, in the absence of an optimal diet, administration of the entire B-vitamin group, rather than a small sub-set, at doses greatly in excess of the current governmental recommendations, would be a rational approach for preserving brain health. ...
    RDAs are population statistics and they therefore represent rough estimates of the average requirement of individuals within a group/population, with an adjustment for the variations in the need for the nutrient among the individuals that make up the population. However, for most micronutrients some of the information that would be required to accurately calculate the daily requirement is either unknown or incomplete, and the recommendations are therefore made on the basis of a number of assumptions and considerations that could lead to large variations in the eventual RDA [81,82]. These figures have also changed little in the last four decades, despite emerging evidence of striking individual differences in the absorption and excretion of vitamins as a consequence of a wide range of factors, including specific genetic polymorphisms, gender, ethnicity, endocrine dysfunction, thyroid function, the habitual co-consumption of medicines, drugs, alcohol and other dietary factors, obesity, overall energy consumption, vigorous exercise, and age [9,21,45,83,84,85,86]. These gaps in our knowledge question the very existence of a “normal” population [87], and suggest that RDAs are, to some extent, arbitrary figures.
    Government figures also show that sizeable minorities of the populations of developed countries fail to consume even the minimum recommended quantity of any given micronutrient. As an example, Troesch et al. [88] presented data showing that a sizeable proportion of the populations of the US and several European countries consume less than the RDA for each of the five B vitamins that they assessed. They note that “a gap exists between vitamin intakes and requirements for a significant proportion of the population”. As a result, studies assessing the blood levels of vitamins show that small but significant proportions of the populations of developed countries have biochemical levels of each of the B vitamins that may well predispose them to deficiency related diseases. ... For instance, recent US government data [91] demonstrated that 10.5% of the entire US population were biochemically deficient in vitamin B6. A subsequent independent analysis that excluded the substantial minority taking supplements containing vitamin B6 demonstrated much higher deficiency rates of between 23% and 27% for adults, depending on age [16].
  3. ^ Bragg DA, Walling A (2015). "Metabolic Syndrome: Hyperlipidemia". FP Essent. 435: 17–23. PMID 26280341. When metabolic syndrome includes lipid abnormalities, management goals are weight loss and cardiovascular risk management through lifestyle modifications (eg, diet, exercise), and, when appropriate, lowering of lipid levels with pharmacotherapy. ... Lipid levels should be reevaluated 4 to 12 weeks after initiating therapy; lipid levels can be measured without fasting. A lack of improvement often indicates nonadherence. Bile acid sequestrants, fibric acids, and niacin can be used if other drugs are not tolerated. 
  4. ^ McCarty MF, DiNicolantonio JJ (2014). "The molecular biology and pathophysiology of vascular calcification". Postgrad Med. 126 (2): 54–64. doi:10.3810/pgm.2014.03.2740. PMID 24685968. Vascular calcification (VC), commonly encountered in renal failure, diabetes, and aging, is associated with a large increase in the risk for cardiovascular events and mortality. Calcification of the arterial media and of heart valves clearly plays a mediating role in this regard, whereas it is less clear how calcification of plaque influences atherogenesis and risk for plaque rupture. Vascular calcification is an active process in which vascular smooth muscle cells (VSMCs) adopt an osteoblastic phenotype and deposit hydroxyapatite crystals; apoptosis of VSMCs also promotes this deposition. Drivers of this phenotypic transition, which include elevated serum phosphate, advanced glycation end-products, bone morphogenetic proteins, inflammatory cytokines, and leptin, invariably induce oxidative stress in VSMCs ... Antioxidants that suppress reduced nicotinamide adenine dinucleotide phosphate oxidase activity may have the potential to block the osteoblastic transition of VSMCs. Minimizing the absorption of dietary phosphate may also be helpful in this regard, particularly in renal failure, and it can be achieved with plant-based dietary choices, avoidance of phosphate additives, and administration of pharmaceutical phosphate binders, supplemental magnesium, and niacin. 

Cognitive control deteriorates (poorer reasoning and problem solving, forgetting things, and impaired ability to exercise discipline and self-control)[1] as a result of:

  • excessive stress[1]
  • sadness/depression[1]
  • social isolation/loneliness[1]
  • poor health[1]
  • lack of physical exercise/fitness[1]

Impaired cognitive control can produce symptoms of ADHD in adults who did not have the disorder as a child.[1]

Cognitive control can be improved[1] through frequent and repeated:

  • aerobic exercise[1]
  • performance of (difficult) executive function training tasks[1]


  1. ^ a b c d e f g h i j Diamond A (2013). "Executive functions". Annu Rev Psychol. 64: 135–168. doi:10.1146/annurev-psych-113011-143750. PMC 4084861free to read. PMID 23020641. Core EFs are inhibition [response inhibition (self-control—resisting temptations and resisting acting impulsively) and interference control (selective attention and cognitive inhibition)], working memory, and cognitive flexibility (including creatively thinking “outside the box,” seeing anything from different perspectives, and quickly and flexibly adapting to changed circumstances). ... EFs and prefrontal cortex are the first to suffer, and suffer disproportionately, if something is not right in your life. They suffer first, and most, if you are stressed (Arnsten 1998, Liston et al. 2009, Oaten & Cheng 2005), sad (Hirt et al. 2008, von Hecker & Meiser 2005), lonely (Baumeister et al. 2002, Cacioppo & Patrick 2008, Campbell et al. 2006, Tun et al. 2012), sleep deprived (Barnes et al. 2012, Huang et al. 2007), or not physically fit (Best 2010, Chaddock et al. 2011, Hillman et al. 2008). Any of these can cause you to appear to have a disorder of EFs, such as ADHD, when you do not. You can see the deleterious effects of stress, sadness, loneliness, and lack of physical health or fitness at the physiological and neuroanatomical level in prefrontal cortex and at the behavioral level in worse EFs (poorer reasoning and problem solving, forgetting things, and impaired ability to exercise discipline and self-control). ...
    EFs can be improved (Diamond & Lee 2011, Klingberg 2010). ... At any age across the life cycle EFs can be improved, including in the elderly and in infants. There has been much work with excellent results on improving EFs in the elderly by improving physical fitness (Erickson & Kramer 2009, Voss et al. 2011) ... Inhibitory control (one of the core EFs) involves being able to control one’s attention, behavior, thoughts, and/or emotions to override a strong internal predisposition or external lure, and instead do what’s more appropriate or needed. Without inhibitory control we would be at the mercy of impulses, old habits of thought or action (conditioned responses), and/or stimuli in the environment that pull us this way or that. Thus, inhibitory control makes it possible for us to change and for us to choose how we react and how we behave rather than being unthinking creatures of habit. It doesn’t make it easy. Indeed, we usually are creatures of habit and our behavior is under the control of environmental stimuli far more than we usually realize, but having the ability to exercise inhibitory control creates the possibility of change and choice.